Abstract
Atopic dermatitis (AD) is a chronic and relapsing inflammatory skin disorder that affects 25% of children and 7-10% of adults and an associated $5.2 billion in annual healthcare costs in the U.S. Although the precise etiology of AD is unclear, it is associated with a complex interaction of epidermal barrier defects, abundant S. aureus skin colonization, and immune dysregulation, including an eosinophilic infiltrate in the skin that correlates with disease severity. However, the role of eosinophils in AD pathogenesis is largely undefined.
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