Abstract
Non-melanoma skin cancer (NMSC) is the most common cancer in Caucasians worldwide, caused by chronic exposure to environmental factors such as ultraviolet (UV) radiation and chemicals. MIF is a pleiotropic cytokine that is described as a pro-tumorigenic factor in various tumour entities. D-DT was recently described as a homolog of MIF that shares many biological activities. The aim of the present study was to assess the pathophysiological role of MIF and D-DT in the development and progression of UVB and chemically induced NMSC.
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