Abstract
In the past decades, the incidence of atopic dermatits (AD) has been increasing in adult population, raising the question of potential environmental triggers. We hypothesized that pollution might be a contributing factor. Bisphenol A (BPA) is an endocrine disruptor with unkown pathophysiological consequences of its repeated skin exposure. We aimed to decipher the effects of topical application of BPA onto human epidermal equivalents (HEEs) generated with keratinocytes isolated from healthy skin biopsies.
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