Abstract
Desmosomes are macromolecular cell junctions that play a critical role in adhesion and resisting mechanical stress in epithelial tissue. They have a complex architecture with transmembrane cadherins constituting the adhesive interface and intracellular proteins organized into plaques coplanar with the plasma membrane and integrated with keratin. Formation and remodeling of these junctions is essential in wound healing and development. However, changes in desmosome architecture during assembly and disassembly are not well characterized.
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