Abstract
The dysregulation of host-microbe interactions has been associated with chronic inflammatory diseases such as atopic dermatitis (AD) and psoriasis. While the pathogenicity of skin microbiota members has been well-characterized, a growing body of evidence suggests that certain commensals possess beneficial properties for the host. To establish the feasibility of new biotherapeutic agent targeting skin inflammatory diseases, we identify that Staphylococcus cohnii is a skin commensal capable of inhibiting skin inflammation.
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