Abstract
Tumor evolution and stable resistance state identification remains the main challenge for successful targeted tumor therapies. Previously our group has identified the distinct cell state trajectories in patient advanced basal cell carcinomas (BCCs) and have identified the molecular basis, surface markers for, reversibility of, and transcription factor driver of basal to squamous cell carcinoma transition (BST). In addition to the basal and differentiated tumor states, other trajectories and stable states may also exist.
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