Abstract
Objective: Premature Ovarian Insufficiency (POI) is a condition that affects approximately 1% of the population. Although chromosomal, genetic, infectious, iatrogenic and autoimmune factors have been linked to the condition, in the majority of women, no underlying cause is found [1]. It is now thought that most cases of ‘idiopathic’ POI may have genetic causation. FOXE1 is a gene that belongs in the family of forkhead transcription factors. Its single exon codes for a poly-alanine tract in the final protein product.
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