Abstract
The review article presents some issues of the etiopathogenesis of the formation of anemic cardiomyopathy. According to various sources, the pathogenetic onset of anemic cardiomyopathy should be considered a decrease in oxygenation or hemic hypoxia, which in the first stages is accompanied by the development of compensatory mechanisms. With the development of severe forms of anemia, systemic vascular resistance and afterload on the left ventricle significantly increase and lead to a decrease in the ejection fraction of the left ventricle, which subsequently leads to the formation of cardiac pathology in the form of “anemic cardiomyopathy”.
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