Abstract

Purpose . To study the anatomical topographic and clinical features and preconditions for the redundant mobility of the posterior capsule in cataract phacoemulsification. Material and methods . The main group I and the comparative group II were formed. The group I included phacoemulsification results with the intraocular lens (IOL) implantation in 12 patients with brown nuclear cataract, the absence of the posterior layer of cortical masses, the increase in fiber length of the ciliary zonule by more than 0.7mm with its local breaks and the presence of the pseudoexfoliation syndrome of different degrees. The group II included 13 patients with brown nuclear cataract, the absence of the posterior cortical layer and the increase in fiber length of the ciliary zonula less than 0.7mm. Patients had no pseudoexfoliation syndrome or its 1st st age. The magnitude of the posterior capsule excursion in absence of posterior epi-nucleus and emulsification of the last fragment of the nucleus was determined intraoperatively in all patients visually and according to the analysis of video r ecording. Results . In the group I the greatest mobility of the posterior capsule was observed in patients with the absence of the posterior fiber portion in the ciliary zonula and their damages in the anterior and medial portions. In 4 cases a pronounced mobility of posterior capsule led to its rupture in a round shape without hyaloid membrane damage. The damage was transferred into a 4mm posterior capsulorhexis and IOL was implanted into the capsular bag. The postoperative visual acuity was 0.68±0.07. In the group II any pathological mobility of the posterior capsule and its ruptures was not detected. The postoperative visual acuity was 0.61±0.07. Conclusion . The symptom of the posterior capsule pathological mobility is formulated and the conditions for its occurrence are determined. The criteria for the ciliary zonule system alteration are studied according to the UBM data in the presence of which the symptom of the posterior capsule pathological mobility occurs.

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