Palm kernel fixed oil (PKO) extracted by heat (HPKO) and cold-press (CPKO) was evaluated for contact toxicity against Callosobruchus chinensis (L.) at 0–1.5 mL/25 g of cowpea (Vigna unguiculata (L.) Walp) seeds and fungistatic potential against Aspergillus species. In addition, biochemical compounds in the oils were obtained using gas chromatography and mass spectrometric (GC-MS) analysis. One hundred percent mortality of C. chinensis was achieved with ≥1 mL HPKO dose levels while the same mortality was achieved with ≥0.75 mL CPKO dose levels at 24, 48, 72 and 96 h post treatment. Zones of inhibitions (cm) of A. niger (0.93 ± 0.07) and A. flavus (1.27 ± 0.15) with HPKO were comparably higher than zones of inhibitions of A. niger (0.20 ± 0.06) and A. flavus (1.13 ± 0.03) with CPKO. The relative abundance of major compounds in HPKO was Lauric acid > caprylic acid > capric acid while 9-Octadecenoic acid -, 2,3-bis[(trimethylsilyl)oxy]propyl ester > Butanoic acid trimethylsilyl ester > Tetradecanoic acid trimethylsilyl ester in CPKO. The higher percentage of lauric acid in HPKO may account for its greater insecticidal and fungistatic potentials.