Pleomorphic Adenoma Of Soft Palate Research Articles

Fine needle aspiration cytology diagnosed pleomorphic adenoma of soft palate- A rare case report

Pleomorphic adenoma (PA) is benign tumor of salivary gland. Pleomorphic adenoma contains both epithelial and myoepithelial elements and it arises from myoepithelial cells of salivary gland. The index study to describe rare case of pleomorphic adenoma of soft palate diagnosed by FNAC (fine needle aspiration cytology).

Fine needle aspiration cytology diagnosed Pleomorphic adenoma of Soft Palate- A rare case report

Pleomorphic adenoma (PA) is benign tumor of salivary gland. Pleomorphic adenoma contains both epithelial and myoepithelial elements and it arises from myoepithelial cells of salivary gland. The index study to describe rare case of pleomorphic adenoma of soft palate diagnosed by FNAC (fine needle aspiration cytology).

Novel Approach for Management of Extremely Rare Pleomorphic Adenoma of Soft Palate (Minor Salivary Glands).

Salivary gland tumours account for 2-3% of tumours occurring in head and neck region, majority of them being benign. Pleomorphic Adenoma present mainly in Parotid gland and Sub-mandibular gland and rarely in minor salivary glands. The tumours of minor salivary glands are generally malignant. The commonest sites of intra oral Pleomorphic adenoma are palate, lip and buccal mucosa. Here I describe a very rare case of Pleomorphic Adenoma in minor salivary glands and its novel management using CO2 Laser. There was no recurrence seen after a follow-up period of around one year.

Pleomorphic adenoma of soft palate with extensive squamous metaplasia – A diagnostic enigma

Pleomorphic adenoma is the most common salivary gland tumor. It can have a wide range of morphological spectrum. But extensive squamous metaplasia mimicking squamous cell carcinoma is quite uncommon. Palate is a common site for minor salivary gland tumor. Less than hundred cases of pleomorphic adenoma with extensive squamous metaplasia have been reported in English literature, thus making it a rare entity. A 60-year old female presented with a well-defined swelling in the soft palate. The histology was reported as pleomorphic adenoma of soft palate with extensive squamous metaplasia and focal adipocytic metaplasia. The histopathological identification of this entity is important as it can mimic malignancy, particularly squamous cell carcinoma. Correct diagnosis helps in unnecessary aggressive therapy.

Pleomorphic Adenoma of Soft Palate - A rare presentation

Pleomorphic adenoma is the most common tumor of salivary glands. Preferentially, 90% of these tumors occur in the parotid gland and the other 10% occur in the minor salivary glands. The common sites for pleomorphic adenoma of the minor salivary glands include palate, lips and cheeks. These sites are later followed by floor of mouth, tongue, tonsil, pharynx, retromolar area and nasal cavity. It usually presents as a slow growing, painless lesion in the mouth. In the present article, we report a case of 30 year-old female with pleomorphic adenoma of the soft palate. The tumor was removed by excision. Histology revealed both epithelial and mesenchymal components which confirmed the diagnosis. No recurrence was noticed during the follow-up period of 10 months.

Carcinoma ex pleomorphic adenoma of soft palate with cavernous sinus invasion

BackgroundCarcinoma ex pleomorphic adenoma (CXPA) is an aggressive salivary gland malignancy and rare in minor salivary gland. A soft palate CXPA initially presenting as direct cavernous sinus (CS) invasion is very rare.Case PresentationA 60-year-old male had a 3-month history of a small soft palatal mass with progressing left cheek numbness, proptosis, and disturbed vision. Biopsy of soft palatal tumor showed pleomorphic adenoma. Magnetic resonance imaging showed a tumor involving left maxilla, and extended from pterygopalatine fossa, inferior orbital fissure to CS. Excision of tumor revealed CXPA. Adjuvant concomitant chemo-radiation therapy (CCRT) was given. The tumor recurred 5 months later in left CS which was re-treated with CCRT. The disease status was stable at 2 years after the diagnosis of CXPA.ConclusionWe present this case to emphasize that patients with symptoms such as facial numbness, proptosis and disturbed vision should be carefully investigated for lesions invading CS by perineural spread.