Partial Brown-Sequard Syndrome Research Articles

Electrocution Cervical Myelopathy Presenting with Partial Brown Sequard Syndrome: A Case Report and Review of Literature

Electrocution Cervical Myelopathy Presenting with Partial Brown Sequard Syndrome: A Case Report and Review of Literature

Cervical posterior spinal artery infarction causing partial Brown-Sequard syndrome.

Cervical posterior spinal artery infarction causing partial Brown-Sequard syndrome.

Spinal artery infarction.

Spinal cord infarctions are rare, and the symptoms vary depending on location and size. One patient presented with severe neck pain and paresis of the left arm. Compression of a cervical nerve root was initially suspected, but the progression of symptoms and MRI findings gradually suggested a different aetiology.

CMV Transverse Myelitis Presenting as Partial Brown-Sequard Syndrome (P2.319)

CMV Transverse Myelitis Presenting as Partial Brown-Sequard Syndrome (P2.319)

An elderly woman with leg weakness

An 85-year-old woman presented to hospital in March 2012 for evaluation of left leg weakness. On the day of admission she had noticed weakness and clumsiness in the left leg...

Type I spinal arteriovenous fistulae exception: A case for reclassification

ObjectiveType I spinal arteriovenous lesions represent dorsal dural arteriovenous fistulae with no spinal artery involvement. We report an exception to this and propose dividing Type I lesions into dorsal and ventral categories. MethodsA 51-year-old patient presented with a partial Brown-Sequard syndrome. An angiogram revealed a spinal arteriovenous fistula, most prominently being fed a radicular artery arising from the right vertebral artery with only ventral venous drainage. ResultsThis feeder was selected and embolized with onyx, however residual fistula persisted and the patient subsequently underwent microsurgical disconnection. At six-month follow-up, patient motor deficits have resolved and difficulty with proprioception is improving. ConclusionType I dural arteriovenous fistulae are associated with dorsal venous drainage. Our case demonstrates an exception to this. Recognizing this exception was crucial, because it allowed for simple microsurgical disconnection. We propose that type I dural arteriovenous fistulae be subdivided into ventral and dorsal based on their drainage pattern. This differentiation is critical because lesions with ventral drainage have traditionally been classified as type IV. These lesions have a different treatment method given involvement of spinal arteries.

Brown-Sequard syndrome associated with Horner’s syndrome following a penetrating drill bit injury to the cervical spine

We report a 41-year-old male who presented with a partial Brown-Sequard syndrome and Horner’s syndrome following a penetrating drill bit injury to his mid cervical spine. As the injury was not a complete hemisection of the spinal cord, the patient presented with ipsilateral motor deficit and hyperesthesia and diminished contralateral fine touch sensation; however, proprioception, vibration and temperature were all initially intact. A cervical CT and MRI scan showed a damaged spinal cord at the C5/6 level with posterior cord compression secondary to haematoma. A decompressive laminectomy and evacuation of the haematoma was performed. Over the following 5 days the patient’s right-sided motor deficit improved daily; however, he developed a contralateral deficit to pain and temperature upon wakening from the operation which did not resolve. The right-sided Horner’s syndrome also persisted.

Spinal decompression sickness presenting as partial brown-sequard syndrome and treated with robotic-assisted body-weight support treadmill training

To describe the rehabilitation outcome of a case of spinal decompression sickness presenting as partial Brown-Sequard syndrome treated with robotic-assisted body-weight support treadmill training. Case report. Type II decompression sickness patients commonly suffer from myelopathy with gait disturbances necessitating rehabilitation. Robotic-assisted body-weight support treadmill training has been shown to improve the rehabilitation outcome of incomplete spinal cord injury. Its usefulness has not been described in decompression sickness myelopathy. Robotic-assisted body-weight support treadmill training was administrated using the Lokomat. Primary outcomes were American Spinal Cord Association scale, Spinal Cord Independence Measurement, Berg Balance Test, and Walking Index for Spinal Cord Injury. The patient was admitted 3 weeks after the diving injury, with severe paraparesis and a T11 sensory neurological level, resembling partial Brown-Sequard syndrome. After 3 months of rehabilitation including 18 Lokomat sessions, American Spinal Cord Association score improved from C to D, Spinal Cord Independence Measurement improved from 50 to 90 out of 100. Berg Balance Test improved from 35 to 43 out of 56 and Walking Index for Spinal Cord Injury improved from 1 to 15 out of 20. Upon discharge he could walk with one crutch for more than 1 km. Robotic-assisted body-weight support treadmill training for spinal decompression sickness rehabilitation might be beneficial.

Brown-Sequard syndrome due to noncompressive disc prolapse and spinal cord infarction

A 36-year-old female presented with acute onset right mid thoracic pain. Over the subsequent 30 min, she developed weakness of the right lower limb and contralateral numbness and paraesthesia. On examination, there was increased tone in the right lower limb with pyramidal type weakness grade 1–2/5, hyperreflexia and an extensor plantar response. Pyramidal function on the left was normal. In relation to her sensory examination, there was a patch of sensory loss to pain on the right at T7. Below this, there was contralateral pain and temperature loss distal to the knee. Proprioception and vibration were lost on the right to the knee. These signs and symptoms were consistent with a partial Brown-Sequard syndrome on the right at T7. …

Posttraumatic spinal cord herniation

We report a 28-year-old woman who presented with a partial Brown-Sequard syndrome 18 months after a fall from a horse. Investigation revealed the cause of her symptoms to be a spinal cord herniation at the level of T6. A review of previously reported cases of posttraumatic spinal cord herniation was undertaken. Six of the cases reported have clear evidence of injury at the site of subsequent herniation; the remaining five cases may be related to trauma or may be spontaneous spinal cord herniation, with an unrelated history of trauma.

Spontaneous spinal epidural hematoma in an untreated hypertensive patient

An insufficiently treated hypertensive woman developed severe neck pain, followed by a partial Brown-Sequard syndrome, due to a spontaneous spinal epidural hematoma compressing the cervical spinal cord at the levels C4–C7. We discuss the diagnostic difficulties in the acute setting and underline the importance of a careful and detailed neurological evaluation in all patients with any kind of acute neurological deficit. Application of modern imaging techniques in such selected acute cases is helpful in order to establish a diagnosis and initiate therapy. Although rare, clinicians should keep spinal epidural hematoma in mind and consider it as a highly uncommon, yet possible, complication of untreated hypertension.

Spontaneous Cervical Epidural Hematoma

Spontaneous cervical epidural hematoma is a rare cause of neck pain. We present the case of a 64-year-old woman who presented to the emergency department with neck pain from a partial Brown-Sequard syndrome secondary to spontaneous cervical epidural hematoma. The prompt recognition of this entity resulted in a favorable outcome. [Willliams JM, Allegra JR: Spontaneous cervical epidural hematoma. Ann Emerg Med June 1994;23:1368-1370.]