Moffitt's Hypothesis Research Articles

Delinquency and peer acceptance in adolescence: a within-person test of Moffitt's hypotheses.

We tested 2 hypotheses derived from Moffitt's (1993) taxonomic theory of antisocial behavior, both of which are central to her explanation for the rise in delinquency during adolescence. We tested whether persistently delinquent individuals become more accepted by their peers during adolescence and whether individuals who abstain from delinquent behavior become less accepted. Participants were 4,359 adolescents from 14 communities in the PROSPER study, which assessed friendship networks and delinquency from 6th (M = 11.8 years) to 9th (M = 15.3 years) grade. We operationalized peer acceptance as number of nominations received (indegree centrality), attractiveness as a friend (adjusted indegree centrality), and network bridging potential (betweenness centrality) and tested the hypotheses with multilevel modeling. Contrary to Moffitt's hypothesis, persistently delinquent youths did not become more accepted between early and middle adolescence, and although abstainers were less accepted in early adolescence, they became more accepted over time. Results were similar for boys and girls; when differences occurred, they provided no support for Moffitt's hypotheses for boys and were opposite of her hypotheses for girls. Sensitivity analyses in which alternative strategies and additional data were used to identify persistently delinquent adolescents produced similar results. We explore the implications of these results for Moffitt's assertions that social mimicry of persistently antisocial adolescents leads to increases in delinquency and that social isolation leads to abstention. (PsycINFO Database Record (c) 2014 APA, all rights reserved).

An empirical examination of adolescence-limited offending: A direct test of Moffitt's maturity gap thesis

Purpose Provide the first direct test of Moffitt's (1993) hypothesis linking the maturity gap with adolescent delinquency. Methods Data were drawn from the National Longitudinal Study of Adolescent Health (Add Health) and a direct measure of the maturity gap was constructed. Negative binomial regression models—survey-corrected to account for the Add Health research design—were estimated. Results Consistent with Moffitt's theory, the results of the analyses revealed that the maturity gap was predictive of minor forms of delinquency and drug use but not of more serious types of offending behaviors for males. Findings were less supportive of Moffitt's hypothesis for females. Conclusions Moffitt's maturity gap thesis is a viable explanation of adolescent delinquency, especially for males. This portion of the theory, which has largely gone unexamined, warrants further inquiry from criminologists.

Race, the Economic Maturity Gap, and Criminal Offending in Young Adulthood

While research has documented that racial and ethnic groups are differentially involved in juvenile and adult crime, little research has examined whether economic and employment well‐being can explain Black and White adolescents' persistence in criminal activity into young adulthood. One potential explanation emerges from Moffitt, who posits an economic maturity gap to explain Blacks' greater persistence in offending in young adulthood. To evaluate this hypothesis, we draw on three waves of data available in the National Longitudinal Survey of Adolescent Health to examine whether economic and employment well‐being in young adulthood can account for the racial gap, and persistence in offending. Findings are consistent with Moffitt's hypothesis and indicate that economic and employment well‐being in young adulthood explain Blacks' greater involvement in criminal and violent offending in young adulthood. In addition, results indicate that the greater tendency of Blacks, compared to Whites, to persist in violent offending is also driven by the reduced economic and employment well‐being that Blacks face in young adulthood.

On the relationship between cognitive abilities and life-course-persistent offending among a sample of African Americans: A longitudinal test of Moffitt's hypothesis

Prior criminological research showed that cognitive abilities were related to delinquent and criminal activity, primarily within adolescent samples. Moffitt's developmental taxonomy anticipates that cognitive abilities will relate to criminal activity differently throughout the population of offenders, mattering more for life-course-persistent than adolescence-limited offenders. Unfortunately, prior research had not examined in great detail the long-term influence of cognitive abilities on criminal activity from birth to adulthood nor had research explored this issue within an African American sample. In this study, data from the Philadelphia portion of the National Collaborative Perinatal Project (NCPP) were used to examine the long-term effect of cognitive abilities on criminal activity from birth to adulthood.

Testing moffitt's neuropsychological variation hypothesis for the prediction of life-course persistent offending

Although neuropsychological deficits have been linked to antisocial behavior, theories of crime have been slow to develop formal processes and predictions for neuropsychological deficits. One recently articulated theory, however, places emphasis on the influence of neuropsychological variation in the development of antisocial behavior. In her developmental taxonomy. Moffitt outlines a theory of antisocial behavior that rests on the presence of two groups of offenders: life-course persisters and adolescent-limiteds. Both groups occupy distinct etiologies for antisocial involvement and are believed to engage in different antisocial activities. One of the main differences between the two groups is the role that neuropsychological deficits play in the production of antisocial behavior. Moffitt hypothesizes that neuropsychological deficits are predictive of antisocial involvement for life-course persisters but not for adolescent-limiteds. Using data from the Philadelphia portion of the National Collaborative Perinatal Project, proxy measures of neuropsychological deficits are used to predict four different manifestations of life-course persistent offending. Results support Moffitt's hypothesis about the relationship between poor neuropsychological test scores and life-course persistent styles of offending.

Infant and Toddler Pathways Leading to Early Externalizing Disorders

To examine pathways leading to specific types of early externalizing disorders. Longitudinal data were collected on 310 low-income, male subjects followed from infancy until age 6 years. Support across informants was found for the importance of the caregiving environment during infancy in relation to the development of externalizing disorders at school entry. Support was also found for the significance of early child factors, but this was limited to child behavior at home. The results are consistent with social learning and attachment models, which suggest that severe conduct problems in early childhood are the result of deficits in the caregiving environment. Support was also found for Moffitt's hypothesis that children with the comorbid attention-deficit/hyperactivity disorder-oppositional defiant disorder/conduct disorder pattern experience multiple child and psychosocial risk factors that begin during infancy.

Adolescence-limited versus persistent delinquency: Extending Moffitt's hypothesis into adulthood.

The authors examined how neuropsychological, personality, and environmental risk factors and their interactions were related to trajectories of delinquent behavior from adolescence to adulthood. Four waves of longitudinal data from 698 male participants, ages 12-18 at Time 1 and ages 25-31 at Time 4, were included in the analyses. Using a growth mixture model approach, 4 trajectories were identified: nondelinquents, adolescence-limited delinquents, adolescence-to-adulthood-persistent delinquents, and escalating delinquents. Five risk factors distinguished escalating from persistent delinquents and 5 also distinguished nondelinquents from the 3 delinquency trajectories. Persistent delinquents scored significantly higher than adolescence-limited delinquents on only one risk factor, disinhibition. Overall, few of the factors that are related to childhood-to-adolescence persistence were associated with persistence in delinquency beyond adolescence.

Adolescence-limited versus persistent delinquency: extending Moffitt's hypothesis into adulthood.

The authors examined how neuropsychological, personality, and environmental risk factors and their interactions were related to trajectories of delinquent behavior from adolescence to adulthood. Four waves of longitudinal data from 698 male participants, ages 12-18 at Time 1 and ages 25-31 at Time 4, were included in the analyses. Using a growth mixture model approach, 4 trajectories were identified: nondelinquents, adolescence-limited delinquents, adolescence-to-adulthood-persistent delinquents, and escalating delinquents. Five risk factors distinguished escalating from persistent delinquents and 5 also distinguished nondelinquents from the 3 delinquency trajectories. Persistent delinquents scored significantly higher than adolescence-limited delinquents on only one risk factor, disinhibition. Overall, few of the factors that are related to childhood-to-adolescence persistence were associated with persistence in delinquency beyond adolescence.

Cognitive abilities in adolescent-limited and life-course-persistent criminal offenders.

T. E. Moffitt's (1993a) hypothesis that adolescent-limited criminal offenders will have higher scores on tests of cognitive ability than life-course-persistent offenders was tested with 12 tests of cognitive ability given to a large and diverse sample of delinquent juveniles whose arrest records were collected over 20 years. This is the first investigation to empirically evaluate this proposal with longitudinal data obtained from a sample for a long enough time to distinguish life course patterns of crime. This study provided only partial support for Moffitt's hypothesis because the results varied by ethnicity. We found relatively consistent support for the hypothesis for Caucasians and Hispanics but no support for the hypothesis for African Americans. These findings are interpreted in terms of differences in developmental contexts for individual ethnic groups.

THE INFLUENCE OF GENDER, LOW BIRTH WEIGHT, AND DISADVANTAGED ENVIRONMENT IN PREDICTING EARLY ONSET OF OFFENDING: A TEST OF MOFFITT'S INTERACTIONAL HYPOTHESIS

Little is known about the causes of an early onset of offending. In an attempt to shed light on this issue, some theoretical models have been advanced purporting to explain the reasons for an individual's early initiation into offending. In one of these models, Moffitt (1993) predicts that early onset of offending is caused by an interaction between (1) increased risk for neuropsychological disorders and (2) disadvantaged childhood environments. This study tests Moffitt's hypothesis concerning the development of early offending. In the present analysis, low birth weight was used as a proxy for increased likelihood of neuropsychological deficits, and socioeconomic status and family structure served as indicators for disadvantaged environment. Using the Philadelphia portion of the Collaborative Perinatal Project, we find support for Moffitt's hypothesis that neuropsychological risk and disadvantaged environment interact to produce an early, but not late, onset of offending. In subsequent analysis, the interaction was observed for males but not females. The latter result, however, may be a function of the small number of cases in the female sample. Finally, we address the theoretical and policy implications arising from our analyses and provide some suggestions for future research.