Spasms Of Masticatory Muscles Research Articles

CT-guided radiofrequency ablation of facial and mandibular nerves in the treatment of compound Meige’s syndrome

This retrospective study examined the clinical outcomes and complications in 6 cases of compound Meige’s syndrome, presenting with blepharospasm and masticatory muscle spasm, following treatment with CT-guided radiofrequency ablation targeting bilateral facial nerves and mandibular branches of the trigeminal nerve. After the operation, the symptoms of eyelid spasm and masticatory muscle spasm resolved, but mild facial paralysis and numbness of mandibular nerve innervation persisted. Follow-up for 4–28 months showed that the symptoms of facial paralysis resolved within 2–5 (3.17 ± 0.94) months after the operation, whereas the numbness in the mandibular region persisted, accompanied by a decrease in masticatory function. During the follow-up period, none of the 6 patients experienced a recurrence of Meige’s syndrome. These findings suggest that CT-guided radiofrequency ablation of the bilateral facial nerve and mandibular branches of the trigeminal nerve may offer a promising approach to treating compound Meige’s syndrome.

Efficacy of CT-guided partial radiofrequency ablation of bilateral responsible cranial nerves in the treatment of Meige syndrome

Objective: To evaluate the efficacy of CT-guided partial radiofrequency ablation of bilateral responsible cranial nerves in the treatment of Meige syndrome. Methods: The Clinical data of 56 patients with Meige syndrome in the Department of Pain Medicine, Affiliated Hospital of Jiaxing University from June 2019 to January 2023 were retrospectively analyzed [19 males and 37 females, aged 42-76 (58.6±8.3) years], including 51 cases of blepharospasm, 3 cases of oromandibular dystonia and 2 cases of blepharospasm concomitant with oromandibular dystonia. CT-guided partial radiofrequency ablation of bilateral responsible cranial nerves was performed on different types of Meige syndrome. And the efficacy and complications of the technique were observed. Results: Fifty-one patients with blepharospasm Meige syndrome underwent CT-guided radiofrequency of facial nerve through bilateral stylomastoid foramen punctures, the symptoms of blepharospasm disappeared completely, leaving bilateral mild and moderate facial paralysis symptoms. Three patients with oral-mandibular dystonia underwent CT-guided radiofrequency therapy by bilateral foramen ovale puncture of mandibular branches of trigeminal nerve, masticatory muscle spasm disappeared, the patients had no difficulty opening the mouth, and the skin numbness in bilateral mandibular nerve innervation area was left. Two cases of Meige syndrome with blepharospasm concomitant with oromandibular dystonia were treated by radiofrequency of facial nerve and mandibular branch of trigeminal nerve, and all symptoms disappeared. The patients were followed up for 1-44 months after the operation, and the symptoms of mild and moderate facial paralysis disappeared at (3.2±0.8) months after the operation, but the numbness did not disappear. Three patients with blepharospasm recurred at the 14, 18 and 22 months after the operation, respectively, while the rest cases did not recur. Conclusions: According to different types of Meige syndrome, CT-guided partial radiofrequency ablation of responsible cranial nerves can effectively treat the corresponding type of Meige syndrome. The complications are only mild and moderate facial paralysis which can be recovered, and/or skin numbness in the mandibular region.

Effectiveness of physiotherapy rehabilitation on hysterical trismus

Trismus, known as difficulty in mouth opening, commonly occurs due to disturbances in the trigeminal nerve resulting in spasm of masticatory muscles. With a literature review on rehabilitation of hysterical trismus, it was observed that there were different electrotherapeutic modalities and exercises which have their individual benefits for trismus. However, there is a lack of a set structured exercise and electrotherapy protocol for the rehabilitation of this condition which may help for better and faster prognosis. A 62-year-old female with hysterical trismus was assessed using dial caliper for mouth opening and was treated using physiotherapeutic approaches. Pre- and postintervention values were recorded. Physiotherapy interventions included hot moist pack therapy, myofascial release, goldfish exercises for the temporomandibular joint (TMJ), TMJ mobilization with distraction, ultrasound, and maintenance of mouth opening using ice cream sticks. Aerospace 200-mm dial caliper was used to measure mouth opening. The present report emphasis on the effect of 2 weeks of physiotherapy intervention using a combination of electrotherapy and exercise has benefited the patient in improving the mouth opening and overall quality of life.

Management of Habitual Temporomandibular Joint Dislocation by Eminectomy

Temporomandibular joint (TMJ) dislocation(luxation) occurs when the condyle moves out of the glenoid fossa upon mouth opening and is locked anteriorly to the articular eminence. The dislocation leads to protective contraction or spasm of masticatory muscles, which reinforces the locked state, with or without pain. Usually, the episodes are triggered by any activities that lead to wide mouth opening such as yawning, laughing, and prolonged, excessive mouth opening during a dental or medical exam. It is a distressing and embarrassing situation for the patient. Repetitive dislocation of TMJ is defined as recurrent or habitual TMJ luxation. There are many treatment modalities available ranging from conservative approach to surgical approach. However, conservative approaches usually offer only a temporary solution and they have significant failure rate. Surgical approaches are available with varying degrees of success. Eminectomy is the most predictable surgical treatment for recurrent TMJ luxation. Herein, we report a case of a 47-year old male patient who was diagnosed with bilateral recurrent TMJ luxation that was successfully treated through bilateral eminectomy.

Masticatory muscle spasm in a non‐japanese patient with satoyoshi syndrome successfully treated with botulinum toxin

A non-Japanese patient with Satoyoshi syndrome is presented. Severe masticatory muscle spasms interfered with feeding, but were successfully treated with botulinum toxin.

Hemifacial atrophy and temporomandibular joint pain-dysfunction syndrome

A case is described of a patient with progressive HA who presented because of left TMJ pain, jaw locking and ipsilateral masticatory muscle spasm. Several reports exist in the literature of masticatory muscle spasm associated with HA. A diagnosis of TMJPDS was made and an occlusal splint was provided which produced resolution of the TMJ pain and jaw locking and significantly reduced the masticatory muscle spasms.

The phantom earache. Temporomandibular joint dysfunction in children.

Temporomandibular joint dysfunction is a benign, relatively uncommon childhood disorder and should be considered in children who have intermittent unilateral otalgia of three to four days' duration and whose audiographic, tympanometric, and clinical otologic examinations reveal normal findings. Most of the patients in our study had undergone orthodontic therapy during the year preceding the onset of temporomandibular joint dysfunction, and the vast majority of them had received orthodontic treatment within two weeks of each episode of otalgia. The diagnosis can be confirmed clinically by reproducing the pain associated with masticatory muscle spasm by palpation of the preauricular areas, intraotic manipulation, and palpation of the pterygoid muscles. Treatment consists of administering acetaminophen, applying hot compresses to the preauricular area, and opening and closing the mouth 30 to 40 times after each compress as an effort to interrupt the muscle spasm.

Diagnosis and reconstruction of the human temporomandibular joint after trauma or internal derangement.

This study reviewed the standardized records of 1100 patients with the symptoms of temporomandibular joint syndrome. Of these patients, only 4.5 percent required surgical intervention. The remaining patients were found to have masticatory muscle spasm and were treated by conservative dental methods. Over half the surgical patients had significant macrotrauma to the jaws in their past history. In addition, electromyographic measurement of the masseteric silent period duration in these patients did not reveal muscle spasm. These factors further serve to differentiate the surgical patient from the patient with myofascial pain dysfunction. The patients selected for surgery demonstrated moderate to severe joint disease and required arthroplasty with partial meniscectomy. A surgical technique is presented demonstrating the reconstruction of the meniscus with silicone implant. This same surgical technique is studied in 10 monkeys, and their joints are examined histologically. The results of surgery reveal that 87 percent of the patients reported improvement 1 year after surgery. In all patients complaining of temporomandibular joint clicking or crepitus, surgery produced complete alleviation of these symptoms. The results of surgery were also associated with a 62 percent increase of jaw opening. Histologic evaluation of the human meniscal resections revealed that in addition to an anatomic displacement of the meniscus, there are also significant cellular changes. These changes consisted of calcification, a decrease in cellularity, hyperemia, and a decrease in elastin content.

Etiology of the pain-dysfunction syndrome

According to the psychophysiologic theory, masticatory muscle spasm is the primary factor in the myofascial pain-dysfunction syndrome. Emotional, rather than mechanical, factors are the prime etiologic agents in stimulating chronic oral habits that produce muscle fatigue. According to the psychophysiologic theory, masticatory muscle spasm is the primary factor in the myofascial pain-dysfunction syndrome. Emotional, rather than mechanical, factors are the prime etiologic agents in stimulating chronic oral habits that produce muscle fatigue.