Effects Of Repeated-sprint Training Research Articles

The Effects of Repeated-Sprint Training on Physical Fitness and Physiological Adaptation in Athletes: A Systematic Review and Meta-Analysis.

Repeated-sprint training (RST) is a common training method for enhancing physical fitness in athletes. To advance RST prescription, it is important to understand the effects of programming variables on physical fitness and physiological adaptation. This study (1) quantifies the pooled effects of running RST on changes in 10 and 20m sprint time, maximal oxygen consumption (VO2max), Yo-Yo Intermittent Recovery Test Level 1 (YYIR1) distance, repeated-sprint ability (RSA), countermovement jump (CMJ) height and change of direction (COD) ability in athletes, and (2) examines the moderating effects of program duration, training frequency, weekly volume, sprint modality, repetition distance, number of repetitions per set and number of sets per session on changes in these outcome measures. Pubmed, SPORTDiscus and Scopus databases were searched for original research articles up to 04 July 2023, investigating RST in healthy, able-bodied athletes, between 14 and 35years of age, and a performance calibre of trained or above. RST interventions were limited to repeated, maximal running (land-based) sprints of ≤ 10s duration, with ≤ 60s recovery, performed for 2-12weeks. A Downs and Black checklist was used to assess the methodological quality of the included studies. Eligible data were analysed using multi-level mixed-effects meta-analysis, with standardised mean changes determined for all outcomes. Standardised effects [Hedges G (G)] were evaluated based on coverage of their confidence (compatibility) intervals (CI) using a strength and conditioning specific reference value of G = 0.25 to declare an improvement (i.e. G > 0.25) or impairment (i.e. G < - 0.25) in outcome measures. Applying the same analysis, the effects of programming variables were then evaluated against a reference RST program, consisting of three sets of 6 × 30m straight-line sprints performed twice per week for 6weeks (1200m weekly volume). 40 publications were included in our investigation, with data from 48 RST groups (541 athletes) and 19 active control groups (213 athletes). Across all studies, the effects of RST were compatible with improvements in VO2max (G 0.56, 90% CI 0.32-0.80), YYIR1 distance (G 0.61, 90% CI 0.43-0.79), RSA decrement (G - 0.61, 90% CI - 0.85 to - 0.37), linear sprint times (10m: G - 0.35, 90% CI - 0.48 to - 0.22; 20m: G - 0.48, 90% CI - 0.69 to - 0.27), RSA average time (G - 0.34, 90% CI - 0.49 to - 0.18), CMJ height (G 0.26, 90% CI 0.13-0.39) and COD ability (G - 0.32, 90% CI - 0.52 to - 0.12). Compared with the reference RST program, the effects of manipulating training frequency (+ 1 session per week), program duration (+ 1 extra training week), RST volume (+ 200m per week), number of reps (+ 2 per set), number of sets per session (+ 1 set) or rep distance (+ 10m per rep) were either non-substantial or comparable with an impairment in at least one outcome measure per programming variable. Running-based RST improves speed, intermittent running performance, VO2max, RSA, COD ability and CMJ height in trained athletes. Performing three sets of 6 × 30m sprints, twice per week for 6weeks is effective for enhancing physical fitness and physiological adaptation. Additionally, since our findings do not provide conclusive support for the manipulation of RST variables, further work is needed to better understand how programming factors can be manipulated to augment training-induced adaptations. Open Science Framework registration https://doi.org/10.17605/OSF.IO/RVNDW .

Repeated-sprint training in hypoxia boosts up team-sport-specific repeated-sprint ability: 2-week vs 5-week training regimen.

To investigate (1) the boosting effects immediately and 4 weeks following 2-week, 6-session repeated-sprint training in hypoxia (RSH2-wk, n = 10) on the ability of team-sport players in performing repeated sprints (RSA) during a team-sport-specific intermittent exercise protocol (RSAIEP) by comparing with normoxic counterpart (CON2-wk, n = 12), and (2) the dose effects of the RSH by comparing the RSA alterations in RSH2-wk with those resulting from a 5-week, 15-session regimen (RSH5-wk, n = 10). Repeated-sprint training protocol consisted of 3 sets, 5 × 5-s all-out sprints on non-motorized treadmill interspersed with 25-s passive recovery under the hypoxia of 13.5% and normoxia, respectively. The within- (pre-, post-, 4-week post-intervention) and between- (RSH2-wk, RSH5-wk, CON2-wk) group differences in the performance of four sets of RSA tests held during the RSAIEP on the same treadmill were assessed. In comparison with pre-intervention, RSA variables, particularly the mean velocity, horizontal force, and power output during the RSAIEP enhanced significantly immediate post RSH in RSH2-wk (5.1-13.7%), while trivially in CON2-wk (2.1-6.2%). Nevertheless, the enhanced RSA in RSH2-wk diminished 4 weeks after the RSH (-3.17-0.37%). For the RSH5-wk, the enhancement of RSA immediately following the 5-week RSH (4.2-16.3%) did not differ from that of RSH2-wk, yet the enhanced RSA was well-maintained 4-week post-RSH (0.12-1.14%). Two-week and five-week RSH regimens could comparably boost up the effects of repeated-sprint training in normoxia, while dose effect detected on the RSA enhancement was minimal. Nevertheless, superior residual effects of the RSH on RSA appear to be associated with prolonged regimen.

Long recovery induced by short exercise-to-rest ratio and effort duration determine the influence of hypoxia during repeated cycling sprints to exhaustion

Introduction&#x0D; Repeated sprints exercise (RSE) performed in hypoxia (RSH) induce greater performance improvement than in normoxia (Brocherie et al., 2017). It has been previously argued that RSH efficiency depend on the oxidative-glycolytic balance which is influenced by sprint duration and exercise-to-rest-ratio (E:R). Indeed, we recently showed that long sprint duration (e.g. 20 s vs 10 s or 5 s) blunts the additional impact of hypoxia on RSH with a constant large E:R of 1:22. However, E:R also influence acute response to RSH. Therefore, this study aims to compare acute responses during RSE to exhaustion with a short E:R (1:6) in normoxia (RSN) and hypoxia (FiO2 = 0.13) and the same sprint durations (5, 10 or 20 s) previously used.&#x0D; Methods&#x0D; On separate visits, 10 active participants completed in random order three RSH and three RSN sessions to exhaustion on a cycle-ergometer (Excalibur Sport, Lode) with three sprint durations and a constant short E:R of 1:6 (5:30; 10:60 and 20:120). Vastus lateralis muscle de-reoxygenation (Oxymon, Artinis Medical Systems) and power output were continuously recorded. Lower limb and breathing discomfort, blood lactate, and ratings of perceived exertion were evaluated immediately after exhaustion.&#x0D; Results&#x0D; Number of sprints and peak power output were higher while blood lactate was lower (all p &lt; 0.001) during 5:30 compared to 10:60 or 20:120. No condition or interaction effects were reported for blood lactate and exercise-related sensation. Blood lactate and muscle deoxyhemoglobin increase (p &lt; 0.001) and total hemoglobin decrease (p = 0.002) during sprint were more pronounced when sprint duration increased (no hypoxia effect).&#x0D; Discussion&#x0D; Similar deoxygenation changes during recovery were reported in hypoxia compared to normoxia suggesting that 1:6 ratio and the associated long recovery period seemed to allow myoglobin O2 store restauration, even with a FiO2 = 13%. Sprint effort should be interspersed by incomplete recovery making impossible the full O2 reloading myoglobin and PCr resynthesis. The increase in blood lactate and the concomitant decrease in muscle oxygenation during sprint with longer duration confirmed a switch in the oxidative-glycolytic balance for energy supply. Since short exercise-to-rest ratio blunt hypoxic effect during RSH due to too long recovery period, hypoxia did not participate to the switch from oxidative to glycolytic energy supply.&#x0D; Conclusion&#x0D; During RSE to exhaustion, a short exercise-to-rest ratio (i.e., 1:6) blunts the specific psychophysiological responses induced by hypoxia, probably due to too long of recovery period. Manipulating effort duration rather than hypoxic exposure is preferable to alter both anaerobic energy system solicitation and power produced during repeated cycling sprints to exhaustion using a short exercise-to-rest ratio.&#x0D; References&#x0D; Brocherie, F., Girard, O., Faiss, R., &amp; Millet, G. P. (2017). Effects of repeated-sprint training in hypoxia on sea-level performance: A meta-analysis. Sports Medicine, 47, 1651-1660. https://doi.org/10.1007/s40279-017-0685-3

Effects Of Repeated-sprint Training In Combined Hot And Hypoxia

Effects Of Repeated-sprint Training In Combined Hot And Hypoxia

Effects of repeated-sprint training in hypoxia induced by voluntary hypoventilation on performance during ice hockey off-season

This study aimed to assess the effects of an off-season period of repeated-sprint training in hypoxia induced by voluntary hypoventilation at low lung volume (RSH-VHL) on off-ice repeated-sprint ability (RSA) in ice hockey players. Thirty-five high-level youth ice hockey players completed 10 sessions of running repeated sprints over a 5-week period, either with RSH-VHL (n = 16) or with unrestricted breathing (RSN, n = 19). Before (Pre) and after (Post) the training period, subjects performed two 40-m single sprints (to obtain the reference velocity (Vref)) followed by a running RSA test (12 × 40 m all-out sprints with departure every 30 s). From Pre to Post, there was no change in Vref or in the maximal velocity reached in the RSA test in both groups. In RSH-VHL, the mean velocity of the RSA test was higher (88.9 ± 5.4 vs. 92.9 ± 3.2% of Vref; p &lt; 0.01) and the percentage decrement score lower (11.1 ± 5.2 vs. 7.1 ± 3.3; p &lt; 0.01) at Post than at Pre whereas no significant change occurred in the RSN group (89.6 ± 3.3 vs. 91.3 ± 1.9% of Vref, p = 0.11; 10.4 ± 3.2 vs. 8.7 ± 2.3%; p = 0.13). In conclusion, five weeks of off-ice RSH-VHL intervention led to a significant 4% improvement in off-ice RSA performance. Based on previous findings showing larger effects after shorter intervention time, the dose-response dependent effect of this innovative approach remains to be investigated.

Distinct Effects of Repeated-Sprint Training in Normobaric Hypoxia and β-Alanine Supplementation

Objective: The present study evaluated the effects of repeated-sprint training in normobaric hypoxia and β-alanine supplementation (BA) on aerobic and anaerobic performance in recreationally active men.Methods: Participants were randomly assigned to one of the following groups: normoxia/β-alanine (NB, n = 11), normoxia/placebo (NP, n = 8), normobaric hypoxia/β-alanine (HB, n = 10) and normobaric hypoxia/placebo (HP, n = 9). All participants completed 8 training sessions over 4 weeks on a cycle ergometer either in normobaric hypoxia (oxygen fraction: FiO2 = 14.2%) or normoxia (FiO2 = 20.9%). Participants were instructed to consume a daily dosage of 6.4 g of BA or placebo. Changes in performance in a graded exercise test, repeated-sprint test (RST), and 3-minute all-out test (3MT) were examined before and after training and supplementation.Results: No between-group differences were observed for training volume or supplementation compliance. Anthropometric and hematological measures remained unchanged before and after intervention in all groups. A main effect of training condition was shown for oxygen consumption and power output at respiratory compensation point, average power output during the last sprint of the RST, heart rate recovery following the RST, and total work during the 3MT. These measures in the normobaric hypoxia groups were significantly (p < 0.05) higher than the normoxia groups, except for the heart rate recovery following the RST. A main effect of supplement was detected in anaerobic working capacity, with postintervention values in the BA groups being significantly (p < 0.05) higher than the placebo groups.Conclusions: Repeated-sprint training in hypoxia improved aerobic performance, exercise tolerance, cardiovascular recovery, and overall working capacity, while BA maintained the anaerobic working capacity. However, BA did not provide additional benefits with respect to attenuating fatigue or enhancing repeated-sprint performance.

Effects of Repeated-Sprint Training in Hypoxia on Tennis-Specific Performance in Well-Trained Players.

This study examined the physiological, physical and technical responses to repeated-sprint training in normobaric hypoxia [RSH, inspired fraction of oxygen (FiO 2 ) 14.5%] vs. normoxia (RSN, FiO 2 20.9%). Within 12 days, eighteen well-trained tennis players (RSH, n=9 vs. RSN, n=9) completed five specific repeated-sprint sessions that consisted of four sets of 5 maximal shuttle-run sprints. Testing sessions included repeated-sprint ability and Test to Exhaustion Specific to Tennis (TEST). TEST’s maximal duration to exhaustion and time to attain the ‘onset of blood lactate accumulation’ at 4 mMol.L −1 (OBLA) improvements were significantly higher in RSH compared to RSN. Change in time to attain OBLA was concomitant with observations similar in time to the second ventilatory threshold. Significant interaction (P=0.003) was found for ball accuracy with greater increase in RSH (+13.8%, P=0.013) vs. RSN (–4.6%, P=0.15). A correlation (r=0.59, P<0.001) was observed between change in ball accuracy and TEST’s time to exhaustion. Greater improvement in some tennis-specific physical and technical parameters was observed after only 5 sessions of RSH vs. RSN in well-trained tennis players.

Effects of Repeated-Sprint Training in Hypoxia on Sea-Level Performance: A Meta-Analysis

Repeated-sprint training in hypoxia (RSH) is a recent intervention regarding which numerous studies have reported effects on sea-level physical performance outcomes that are debated. No previous study has performed a meta-analysis of the effects of RSH. We systematically reviewed the literature and meta-analyzed the effects of RSH versus repeated-sprint training in normoxia (RSN) on key components of sea-level physical performance, i.e., best and mean (all sprint) performance during repeated-sprint exercise and aerobic capacity (i.e., maximal oxygen uptake [[Formula: see text]]). The PubMed/MEDLINE, SportDiscus®, ProQuest, and Web of Science online databases were searched for original articles-published up to July 2016-assessing changes in physical performance following RSH and RSN. The meta-analysis was conducted to determine the standardized mean difference (SMD) between the effects of RSH and RSN on sea-level performance outcomes. After systematic review, nine controlled studies were selected, including a total of 202 individuals (mean age 22.6±6.1years; 180 males). After data pooling, mean performance during repeated sprints (SMD=0.46, 95% confidence interval [CI] -0.02 to 0.93; P=0.05) was further enhanced with RSH when compared with RSN. Although non-significant, additional benefits were also observed for best repeated-sprint performance (SMD=0.31, 95% CI -0.03 to 0.89; P=0.30) and [Formula: see text] (SMD=0.18, 95% CI -0.25 to 0.61; P=0.41). Based on current scientific literature, RSH induces greater improvement for mean repeated-sprint performance during sea-level repeated sprinting than RSN. The additional benefit observed for best repeated-sprint performance and [Formula: see text] for RSH versus RSN was not significantly different.