Effect Of Exercise-induced Stress Research Articles

The accumulated effects of exercise-induced stress in male athletes during a national Ultimate Frisbee competition across four days

Introduction: Ultimate Frisbee (UF) is a field-based team-sport, involving high intensity activities such as jumping, sprinting, acceleration, deceleration and change-of-direction with short recovery periods. Whilst a number of studies have reported the physical demands of an UF match, the level of exercise-induced stress following a match is yet to be determined, particularly during a competition. Understanding the level of stress induced by an UF match may improve recovery strategies and optimise athlete preparedness during an UF competition involving multiple matches across days. Thus, the purpose of this study was to examine the accumulated effects of exercise-induced stress during a national UF competition across four days in male athletes. Methods Twelve elite male UF players (age 29 ± 2 years; height 179.6 ± 5.1 cm; and body mass 80.0 ± 6.0 kg) who competed in the Australian Men's National UF competition in 2019 volunteered for this study. The competition was held across four consecutive days (D1, D2, D3 and D4), with athletes undertaking 1-3 matches per day. Every morning (08:00-12:00), within the hour prior to the first match, the following assessments were undertaken for each athlete: delayed onset of muscle soreness (DOMS) using a 1-5 Likert scale (‘no soreness’ to ‘very sore’), leg power during a drop-jump from a 30 cm box, and speed during a 10m sprint. During the drop-jump protocol, athletes jumped on a force plate with a dowel on their shoulder that hosted an accelerometer. Peak velocity and muscular power was derived from the accelerometer, whilst jump height, contact time and reactive strength index (RSI: flight time/contact time) were quantified from the force plate. Results: When compared to D1, the level of DOMS was significantly increased on D2, D3 and D4. Conversely, peak jump velocity and jump height were significantly decreased on D2 and D4 while RSI and muscular power were significantly decreased on D3 and D4. No significant differences were identified across days for contact time and 10m sprint time. Discussion: In the current study, multiple UF matches performed in a single day induced stress sufficient for DOMS and impair neuromuscular performance during a drop jump task on subsequent days. In addition, an accumulation of exercise-induced stress throughout the UF competition was observed with DOMS peaking and the majority of drop-jump performance measures impaired during the last day. Thus, appropriate recovery strategies should be implemented to minimise carry-over effects of exercise-induced stress across days of an UF competition.

P23 Assessing the effects of exercise-induced stress on the Fiona mouse model

P23 Assessing the effects of exercise-induced stress on the Fiona mouse model

P22 Clinical and pathological heterogeneity in partial merosin deficiency

P22 Clinical and pathological heterogeneity in partial merosin deficiency

Effects of exercise-induced stress and dexamethasone on plasma hormone and glucose concentrations and sedation in dogs treated with dexmedetomidine

To compare the effects of pretreatment with dexamethasone, physical stress (exercise), or both on sedation and plasma hormone and glucose concentrations in dogs treated with dexmedetomidine (DEX). 6 healthy purpose-bred Beagles. Dogs received 4 treatments each in a randomized order prior to i.v. administration of DEX (5 fLg/kg). Pretreatments were as follows: (1) i.v. administration of saline (0.9% NaCI) solution and no exercise (control group); (2) IV administration of dexamethasone (0.05 mg/kg) and no exercise (DM group); (3) i.v. administration of saline solution and exercise (EX group; 15 minutes of trotting on a treadmill at a speed of 2 m/s); and (4) i.v. administration of dexamethasone and exercise (DM+EX group). Following DEX administration, all dogs had similar times to recumbency and sedation index values, irrespective of pretreatment with values, irrespective of pretreatment with dexam-d ethasone or exercise. Plasma catecholamine concentrations decreased after DEX administration. Compared with control group dogs, plasma cortisol concentrations were higher in EX-group dogs prior to DEX administration and lower in DM- and DM+EX-group dogs following DEX administration. Administration of DEX decreased plasma cortisol concentration in EX-group dogs only. Plasma glucose concentration was not influenced by exercise or dexamethasone administration was lower than baseline concentrations at 30 minutes after DEX administration and returned to baseline values by 90 minutes. Heart and respiratory rates and rectal temperature increased during exercise. After DEX administration, these values decreased below baseline values. The decrease in heart rate was of shorter duration in dogs that underwent pretreatment with dexamethasone, exercise, or both than in control group dogs. Pretreatment with dexamethasone, moderate physical stress (exercise), or both did not influence sedation or cause adverse effects in healthy dogs treated with DEX.

The influence of combined treatment with propranolol and acetylsalicylic acid on platelet aggregation in coronary heart disease.

British Journal of Clinical PharmacologyVolume 7, Issue 3 p. 287-291 Free Access The influence of combined treatment with propranolol and acetylsalicylic acid on platelet aggregation in coronary heart disease. I Keber, I KeberSearch for more papers by this authorM Jerse, M JerseSearch for more papers by this authorD Keber, D KeberSearch for more papers by this authorM Stegnar, M StegnarSearch for more papers by this author I Keber, I KeberSearch for more papers by this authorM Jerse, M JerseSearch for more papers by this authorD Keber, D KeberSearch for more papers by this authorM Stegnar, M StegnarSearch for more papers by this author First published: March 1979 https://doi.org/10.1111/j.1365-2125.1979.tb00934.xCitations: 16AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat References Abdulla, Y.H. (1969). Beta-adrenergic receptors in human platelets. J. Atheroscler. Res., 9, 171– 179. Born, G.V.R. (1962). Aggregation of blood platelets by adenosine diphosphate and its reversal. Nature, 194, 927– 929. Breddin, K. & Banke, J. (1965). Thrombozyten-agglutination und Gefäskrankheiten. Blut, 9, 144– 164. Deykin, D. (1974). Emerging concepts of platelet function. N. Engl. J. Med., 290, 144– 151. Frishman, W.H., Weksler, B.B., Christodoulou, J.P., Smithen, C. & Killip, T. (1974). Reversal of abnormal platelet aggregability and change in exercise tolerance in patients with angina pectoris following oral propranolol. Circulation, 50, 887– 896. Green, K.G. (1977). Reduction in mortality after myocardial infarction with long-term β-adrenoreceptor blockade. Multicentre international study, supplementary report Br. med. J., 2, 419– 421. Goldenfarb, P.B., Cathey, M.H. & Cooper, G.R. (1970). 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Effects of exercise-induced stress on platelet aggregation.

In order to determine if the stress of a standard exercise test will affect the propensity for platelets to aggregate, platelet aggregometer studies were performed before and after a double two-step Master's test in 19 patients with angina and in 19 normal controls. The response of platelet-rich plasma to 0.1, 1 and 10 muM of ADP and 1 and 10 muM of epinephrine were tested in a Chrono-Log aggregometer. Prior to exercise there was no significant difference between the number of patients and controls responding with irreversible aggregation to any of the five tests. Immediately following exercise, no significant differences in the number of irreversible aggregation responses were seen in the control group. In the group with coronary artery disease, the incidence of irreversible responses was significantly higher after exercise on test with 1 muM ADP (p less than 0.05), 1 muM epinephrine (p=0.02), and 10 muM epinephrine (p less than 0.05). It is concluded that exercise increases significantly the tendency of platelets to aggregate among patients with coronary artery disease. It is possible that this effect may play a role in myocardial infarction induced during stress.