Congenital Sinus Of Valsalva Aneurysm Research Articles

Surgical repair of a ruptured congenital sinus of Valsalva aneurysm: 10-year experience with 286 cases.

Surgical intervention is the main treatment for a ruptured congenital sinus of Valsalva aneurysm (SVA). However, reports on the surgical experience are scarce. We retrospectively analysed the cases of our centre to summarize our 10-year experience. A total of 286 patients who were diagnosed with a congenital ruptured SVA and underwent surgical repair between 2007 and 2016 were identified for the analysis. Follow-up data (mean ± standard deviation: 49.6 ± 34.9 months) were obtained from outpatient department records and telephone calls. The SVAs originated from the right coronary sinus (79.7%), the non-coronary sinus (19.6%) and the left coronary sinus (0.7%) but ruptured into the right ventricle (58.4%) and the right atrium (41.3%). The most commonly associated deformities were a ventricular septal defect (46.3%), aortic valve regurgitation (33.2%) and tricuspid regurgitation (20.3%). The SVA defect was closed by direct suturing (9.1%) or patching (90.9%) through an incision in the cardiac chamber involved or a transaortic approach. The mean postoperative hospital stay duration was 7.2 days, and 98.6% of the patients were discharged in New York Heart Association functional class I or II. The incidence rate of short-term complications was 5.7%. There were 4 late deaths, and 9 patients required rehospitalization due to surgery-related events. The estimated 10-year survival rate was greater than 90% according to the Kaplan-Meier survival curve. Surgical repair is an effective and safe treatment for a ruptured SVA. The majority of patients who undergo surgical repair can survive for a long time.

Progression of Aortic Regurgitation in Asian Patients with Congenital Sinus of Valsalva Aneurysm

We reviewed the experience of An Zhen and Fu Wai Hospital for congenital sinus of Valsalva aneurysm (SVA) to determine risk factors for aortic valve replacement (AVR) and postoperative progression of aortic regurgitation (AR). Over a 7-year period, 255 patients underwent surgical repair of an SVA. Aneurysms originated from the right sinus and the noncoronary sinus in 212 patients (83.1%) and 38 patients (14.9%), respectively, and protruded into the right ventricle and right atrium in 171 patients (67.1%) and 80 patients (31.4%), respectively. AR presented in 142 patients (55.7%), 60 patients underwent AVR, and 13 patients underwent aortic valvuloplasty (3 patients eventually received AVR for valvuloplasty failure). All patients survived the operation. Late death occurred in 2 patients (0.8%), and 2 patients (0.8%) experienced anticoagulation-related complications. Logistic regression analysis revealed that infective endocarditis, the cardiothoracic ratio, and a nonruptured SVA were risk factors for AVR. Late follow-up of 150 patients by echocardiographic assessment revealed that AR improved in 17 patients and worsened in 20 patients. Cox regression analysis revealed AR at discharge to be an independent risk factor for AR aggravation at late follow-up. SVA can be repaired with low mortality and excellent long-term results. AR at discharge is an important factor in determining AR aggravation at late follow-up after the operation. We recommend early diagnosis and aggressive treatment for SVA.

Surgical repair of a sinus of a Valsalva aneurysm ruptured into the left ventricle

A 47-year-old man presented with orthopnea. He had no infection or, in particular, infectious endocarditis. An echocardiogram and multislice computed tomography scan revealed a left sinus of Valsalva aneurysm (SVA) that had ruptured into the left ventricle, with severe aortic regurgitation. He was diagnosed with ruptured congenital SVA with severe aortic regurgitation and underwent closure of the aneurysmal mouth with a Dacron patch, and concomitant aortic valve replacement. The use of a Dacron patch to close the aneurysmal mouth of a ruptured congenital left SVA has not been reported previously in the medical literature.

1210: Ruptured Congenital Sinus of Valsalva Aneurysm Complicated with Congestive Heart Failure in Real-Time Echocardiography

1210: Ruptured Congenital Sinus of Valsalva Aneurysm Complicated with Congestive Heart Failure in Real-Time Echocardiography

Huge Aneurysm of the Sinus of Valsalva Compressing the Left Atrium

Introduction A sinus of Valsalva aneurysm (SVA) can be congenital or acquired. A congenital SVA is more common and most often caused by weakness at the junction of the aortic media and the annulus fibrosus. An acquired SVA is caused by secondary degeneration of the elastic connective tissue that occurs from atherosclerosis or infection (bacterial endocarditis, syphilis, and tuberculosis). Unruptured SVAs usually remain asymptomatic, but sometimes can cause cardiac rhythm abnormalities, myocardial ischemia, and systemic embolic events.

Images in cardiology: Ruptured congenital sinus of valsalva aneurysm

Images in cardiology: Ruptured congenital sinus of valsalva aneurysm

Ruptured sinus of Valsalva aneurysm complicated by myocardial ischemia: pathogenetic mechanisms

Significant left-to-right shunt in combination with severe aortic regurgitation (AR) accelerates the development of symptoms after rupture of congenital sinus of Valsalva aneurysm (SVA) in spite of intact coronary arteries. We depict a rare description of a situation where acute coronary syndrome was the first manifestation of such an occurrence. We believe that the progress of the myocardial ischemia after ruptured SVA depends on the severity of AR and the quantity of the left-to-right shunt. Prompt recognition and surgical repair are indicated to prevent complications of myocardial infarction.

Ruptured Congenital Sinus of Valsalva Aneurysm

Robert L. Smith, M.D.,∗ Anand Irimpen, M.D.,∗ Frederick R. Helmcke, M.D.,† and Edmund Kenneth Kerut, M.D.‡§ ∗Division of Cardiology, Tulane University School of Medicine, New Orleans, Louisiana, †Department of Medicine, Section of Cardiology, LSU Health Sciences Center, New Orleans, Louisiana, ‡Departments of Pharmacology and Physiology, LSU Health Sciences Center, New Orleans, Louisiana, and ‡§Heart Clinic of Louisiana, Marrero, Louisiana

Anesthetic implications of a ruptured aneurysm of the sinus of valsalva

C ONGENITAL SINUS of Valsalva aneurysm (CSVA) is a rare occurrence that accounts for 0.1% to 3.5% of all congenital heart defects.’ The hemodynamic consequences and survival following rupture of a CSVA depend on the severity of heart failure, which may be rapidly progressive. It is important to establish early diagnosis and differentiate this entity from other noncyanotic heart defects, accompanied by a systolic-diastolic murmur, such as patent ductus arteriosus, aorto-pulmonary fistula. or ventricular septal defect (VSD) with aortic insufficiency. There is little information in the literature on the anesthetic considerations of the patient with a ruptured CSVA; therefore, the objective of this report is to emphasize the clinical manifestations and management of these patients in the perioperative period.

Unruptured sinus of Valsalva aneurysm

An unruptured congenital sinus of Valsalva aneurysm (behind the right aortic valve cusp) is described as an incidental necropsy finding in an 82 year old man. Review of previous reports on aneurysms involving only one of the three aortic sinuses discloses that few cases have been described, and that these lesions are rarely diagnosed during life. It is probable, however, that unruptured aortic sinus aneurysm (involving only one sinus) is more common than previous reports indicate, but that, among patients with congenital sinus aneurysm, rupture is likely to occur.