Complication Of Mitral Stenosis Research Articles

Acute aorto-iliac Artery Occlusion: a Rare Complication of Mitral Stenosis

A 42 year old man, diagnosed with rheumatic heart disease and mitral stenosis, presented to the emergency department with acute onset low abdominal pain, severe pain in both lower limbs with paraplegia. The pulses of his feet could not be felt on either side. CT angiogram revealed saddle thrombus at the aorto-iliac junction occluding both common iliac arteries. An intra-arterial thrombolysis was done which was followed by anticoagulation. Within few hours of the thrombolysis, his abdominal pain and pain in the lower limbs subsided and he regained full function of both lower limbs. Pulses in the right foot could be felt but the left posterior tibial pulse was still weak after the procedure. He went on to develop gangrene of the left 2 nd and 3 rd toes which were amputated later. Key words: Acute, Aorto-iliac, Mitral stenosis, Thrombosis.

STUDY ON THE HEART DYSRHYTHM BY 24 HOURS ECG HOLTER IN PATIENTS WITH MITRAL STENOSIS

Objectives: Mitral stenosis is a common disease accounting for 40.3% of population of cardiovascular disease. Cardiac arrhythmia is a common complication of this disease. ECG is an important mean of diagnosing cardiac arrhythmia, especially ECG Holter. The advantage of ECG Holter had provided us with accurate parameters at various moments, both day and night, under exercise and at rest. Therefore, we conduct this research with 2 following objectives: 1. Determine the rate of cardiac arrhythmias and their classification in mitral valve disease throughout ECG Holter. 2. Find out the correlation between cardiac arrhythmia and mitral stenosis severity. Materials and methods: 45 patients (the youngest was 22, the oldest was 73 and average age: 43.84 ± 12.97) were diagnosed with affirmation mitral stenosis based on clinical features, X-ray, and echocardiography. ECG Holter then was recorded and analyzed in the terms of the rate and classification of cardiac arrhythmias. The results showed that: cardiac arrhythmias accounted for 53.33%, in which ventricular premature beats: 88.89%, atrial premature beats: 4.44%, sinus bradycardia: 6.67%, sinus tachycardia: 46.67%, sinus arrest: 11.11%, atrial fibrillation: 55.56%, supraventricular arrhythmias: 2.22%. The more severe mitral stenosis is, the more often heart dysrhythms appear: mitral stenosis severity I, II, III has the rate of heart dysrhythm of 6.67%, 15.56%, 31.10%, respectively. There was an inverse correlation between the severity of mitral stenosis and atrial fibrillation (r=-0.410, p<0.05); inverse correlation between the pulmonary artery pressure and degree of mitral stenosis (r=0,7361, p<0,05). There was a proportional correlation between the pulmonary hypertension and atrial fibrillation (r=0.43, p<0.05). Conclusion: ECG Holter is helpful in diagnosis, follow-up, prognosis of mitral stenosis. It is also valuable in detecting complications of mitral stenosis, aiding in treatment and improving patient’s quality of life.

A complication of mitral stenosis: free floating ball thrombus in left atrium

Left atrial (LA) ball thrombus is a rare disorder. These ball thrombi are generally associated with mitral stenosis, and the restricted mitral orifice encloses the free-floating thrombus in the LA. There is a potential for fatal systemic emboli or mitral valve orifice occlusion that may result in sudden death. We report a rare case of free-floating ball thrombus (FFBT) in the left atrium with mitral stenosis. A 52-year-old woman with chronic mitral stenosis was admitted to the hospital for progressive dyspnea, palpitation and unconsciousness. Electrocardiography showed atrial fibrillation (AF). Echocardiography revealed rheumatic, thickened, and stenotic mitral valve, and FFBT in the left atrium. She was treated successfully with removal of ball thrombus and mitral valve replacement. The size of the ball thrombus was 30×25×15 mm and postoperative course was uneventful.

A Complication of Mitral Stenosis: Free Floating Ball Thrombus in Left Atrium

A Complication of Mitral Stenosis: Free Floating Ball Thrombus in Left Atrium

A complication of mitral stenosis: free floating ball thrombus in left atrium

Left atrial (LA) ball thrombus is a rare disorder. These ball thrombi are generally associated with mitral stenosis, and the restricted mitral orifice encloses the free-floating thrombus in the LA. There is a potential for fatal systemic emboli or mitral valve orifice occlusion that may result in sudden death. We report a rare case of free-floating ball thrombus (FFBT) in the left atrium with mitral stenosis. A 52-year-old woman with chronic mitral stenosis was admitted to the hospital for progressive dyspnea, palpitation and unconsciousness. Electrocardiography showed atrial fibrillation (AF). Echocardiography revealed rheumatic, thickened, and stenotic mitral valve, and FFBT in the left atrium. She was treated successfully with removal of ball thrombus and mitral valve replacement. The size of the ball thrombus was 30×25×15 mm and postoperative course was uneventful.

Aortic embolism complicating mitral stenosis; with particular reference to emergency valvulotomy before embolectomy.

AMONG the most serious complications of mitral stenosis is the formation of a thrombus in the left atrium and the discharge of the thrombus or a part of it as an embolus into the arterial circulation. When, in the presence of mitral stenosis, an embolus lodges at the aortic bifurcation the state of compensation of the heart is of major importance in the survival of the patient after embolectomy. When there is severe cardiac decompensation, the risk of operation for removal of the aortic embolus may be so great as to contraindicate operation. At the same time, the failure to . . .

PARALYSIS OF THE LEFT RECURRENT LARYNGEAL NERVE IN MITRAL STENOSIS

During the past twenty-five years a rather uncommon complication of mitral stenosis, paralysis of the left recurrent laryngeal nerve, has caused considerable interest. The chief importance of the condition lies, perhaps, in the diagnosis, more especially in its distinction from aortic aneurysm; but in addition there has been a great deal of controversy and discussion as to the method of production of the paralysis and its course and termination. Since Ortner first described the condition (1897), four different theories and many modifications have been proposed as to its mechanism, and each has for its support not only the opinion of various clinical investigators, but also the findings in a number of necropsies. Seventy-four cases have been reported, in seventeen of which necropsy was performed. It is my purpose to report a case diagnosed, treated and examined postmortem in the Montreal General Hospital; to review the literature; to discuss the diagnosis,