Abstract. One of the most common forms of dementia is oestrus dementia, which is primarily characterized by cognitive subversion and neural trance. The most obvious pathology is the strong metal disc and neurogenetic fibbers. In recent years, many studies have shown that the intestinal flora in with the change of the AD's development also brings certain effect, play some role. Our bodies are home to many microorganisms, as well as many other fungi (bacteria, fungi, arthritis, and viruses). They can act either as a host or as a neutral or pathological part of the body, taking on both physical and pathological functions. Among them, 95% of microorganisms inhabit the gastrointestinal tract in an intricate ecological colony state, collectively known as the intestinal microbiota. Gut microbiota can affect the blood-brain barrier (BBB) and neurogenesis. If it is more severe, it can lead to neuroinflammation. Sterile study in mice shows that lack of gut microbiota mice increased permeability of blood brain barrier and brain function has changed. Additionally, gut microbiome-targeted therapies, such as GV-971, have shown promise in animal models by modifying gut microbiota composition, reducing neuroinflammation, and improving cognitive function. These findings suggest that modulating the gut microbiome could offer novel therapeutic strategies for AD This emphasizes the need for further research in this area to develop effective treatments.
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