Axillary Artery Aneurysm Research Articles

Rare locations of peripheral aneurysms in Marfan syndrome treated surgically: a case report

Marfan syndrome (MFS) is a connective tissue disorder that can lead to cardiovascular and musculoskeletal abnormalities. Aortic aneurysms and dissections are frequently seen in patients with MFS whereas peripheral vascular aneurysms in subclavian and axillary arteries territory considered very unusual. We reported a case of 54-year-old female with known history of MFS who had undergone a mechanical valve Bentall procedure due to severe aortic regurgitation and ascending aorta aneurysm in addition to thoracoabdominal aortic repair and who presented with a pulsatile painful mass in her right axillary region that turn to be significant true aneurysms of right subclavian and axillary arteries. To relive symptoms and to avoid further complications patient underwent successful surgical repair. Our case demonstrated rare locations of true peripheral aneurysms as a possible manifestation of MFS appeared several years post Bentall procedure and thoracoabdominal aortic repair and highlights also the importance of long-term monitoring to detect earlier such manifestation and avoid complications by surgical repair.

Is there an association between arteriovenous fistulas and axillary artery aneurysms? A report of two cases

IntroductionAxillary artery aneurysms are rare vascular conditions that can present with various clinical manifestations, including neurological deficits and vascular compromise. While the underlying pathophysiology remains complex and multifactorial, potential associations with trauma, arteriovenous fistula formation, and atherosclerosis have been reported.Presentation Of Case.Two male patients, aged 33 and 38, with a history of kidney transplantation and previous arteriovenous fistula (AVF) presented with symptoms of upper limb ischemia and neurological compromise. Imaging revealed large axillary artery aneurysms. Open surgical repair was performed for both cases. Two weeks after discharge, one patient showed good pronation and supination with mildly limited extension. The other patient's wrist drop gradually improved with physiotherapy. DiscussionMultifactorial pathophysiology encompassed altered blood flow dynamics, inflammation, and the underlying vascular pathology. Chief complaints and prior vascular interventions contributed. Open surgical repair was preferred to endovascular approaches, achieving favorable outcomes. ConclusionAxillary artery aneurysms in patients with a history of AVF are rare but potentially serious complications. The cases highlight the complexity of axillary artery aneurysms and the need for careful evaluation and surgical intervention This strategy is crucial to prevent potential complications and optimize patient outcomes. Further research is needed to elucidate the precise pathophysiology and the potential association between AVF and the subsequent development of axillary artery aneurysms. Increasing awareness among surgeons could enable earlier detection of aneurysms, thereby reducing the risk of complications.

Rare Presentations of Takayasu Arteritis: A Case Series

AbstractTakayasu arteritis (TA) is a form of large vessel vasculitis that may lead to fibrosis, stenosis, or aneurysm formation of vessels. Its presentation varies depending on the arterial beds involved. We report 3 cases out of around 150 cases of TA with rare initial presentations of brachial plexopathy caused by an axillary artery aneurysm, complicated type A intramural hematoma, and renal artery aneurysms along with abdominothoracic TA presenting as a pulsatile abdominal mass.

Axillary and brachial artery aneurysm associated with a longstanding arteriovenous fistula

Upper extremity true arterial aneurysms are rare and often present as a pulsatile mass with associated pain and paresthesia from peripheral nerve compression, and can lead to acute limb ischemia from thromboembolic sequela. History of arteriovenous dialysis access and immunosuppression after renal transplantation may contribute to the development of true aneurysms.This report describes the management of a patient with end-stage renal disease (ESRD) with multiple left upper extremity dialysis access who presented with an axillary and brachial artery aneurysm.

Axillary artery aneurysms in pediatric patients: A narrative review

Objectives Axillary artery aneurysms in children are rare and multifactorial. The presenting clinical picture can range from an incidental discovery to threatening symptoms, including loss of extremity function. With the present study we aim to give a comprehensive review of the existing literature focusing on the etiology and management of this rare but threatening pathology. Method A comprehensive review was made in a multi-staged manner. All identified studies have been categorized based on the type of lesions (true or false aneurysm) and their etiologies. Results The treatments differ from the extension of the lesions and from the etiology. Conclusions Open surgery repair with great saphenous vein is still the best long-term approach, but even endovascular, embolization, or conservative management can be considered.

Prevalence of Axillary Artery Variants and Their Clinical Significance: A Scoping Review.

Axillary artery (AA) variants occurred quite commonly, presenting clinical implications. A literature search yielded 523 resultsfrom which 13 parameters were extracted. Some of the AA variants found were the fusion of two or more branches into common trunks, like the fusion of anterior and posterior circumflex humeral arteries. Moreover, several branches were found to emerge from different points than expected, like the lateral thoracic artery's origin from the subscapular artery instead of the second part of the AA. The importance of the knowledge of the AA variations in clinical practice is undeniable and very useful when planning interventional procedures, as in the case of AA aneurysm treatment or in cases of fracture of the surgical neck of the humerus. The heterogeneity of data limited the possibility of a quantitative summary of data. Therefore, a more systemic study of AA variants based on the origin, course, and branching pattern is suggested. The aim of the current review is to summarize current data literatureregarding the AA typical anatomy and its variants, with a focus on their prevalence and possible clinical implications.

Hybrid aortobrachial bypass for a giant subclavian and axillary artery aneurysm in a Marfan patient

Hybrid aortobrachial bypass for a giant subclavian and axillary artery aneurysm in a Marfan patient

WTP1.12 Axillary Artery Aneurysm in a Patient of Achondroplasia

Abstract Background Axillary artery aneurysms are extremely rare mainly occurring as pseudo-aneurysms. They are most commonly caused by trauma however; fungal infections, atherosclerosis, connective tissue diseases, vasculitis and thoracic outlet syndrome are also among the etiological factors. Case Summary Seventeen years old, unmarried, achondroplasic (three feet height) girl presented to OPD with pulsatile swelling and pian in right axilla for six months. Pain was accompanied by weakness and numbness of hand. The swelling was initially small in size which increased over time. Patient had history of left axillary artery aneurysm back in 2015 for which left axillary repair was done. There was no history of axillary trauma. On physical examination an approximately 4×2 cm lump was palpable in right axilla. It was round in shape, mobile, pulsatile and non-expanding with no overlying skin changes. The patient had hypotonia with power of 3/5 in all limbs. Deep tendon reflexes were reduced. Rest of systemic examination was unremarkable. CT angiography was done which showed brachiocephalic trunck and right proximal subclavian artery normal. There was a lobulated enhancing area along the course of the Right axillary artery measuring 3.5×4.7×3.0 cm. The axillary artery aneurysm was excised and arterial continuity was restored by placement of Reverse Saphenous vein graft. Discussion The conventional method of axillary artery aneurysm repair is aneurysmectomy followed by placement of bypass graft. Owing to long term patency, saphenous vein graft serves as an ideal for reconstruction. Conclusion axillary artery aneurysms should be considered as a differential for pulsatile axillary swellings and should be promptly treated to avoid life threatening complications.

Hybrid Aortobrachial Bypass for Giant Subclavian and Axillary Artery Aneurysm in a Patient With Marfan Syndrome

This video demonstrates a challenging case of giant subclavian and axillary artery aneurysm in a patient with Marfan syndrome with multiple prior aortic interventions and coronary artery bypass graft presenting with nerve compression. This patient was successfully treated via a hybrid approach due to complexity. eyJraWQiOiI4ZjUxYWNhY2IzYjhiNjNlNzFlYmIzYWFmYTU5NmZmYyIsImFsZyI6IlJTMjU2In0.eyJzdWIiOiI2M2RkMDAzODFmMmQ3OTliOTkyZTk2MjA0YTEyOTA0MCIsImtpZCI6IjhmNTFhY2FjYjNiOGI2M2U3MWViYjNhYWZhNTk2ZmZjIiwiZXhwIjoxNjg0ODU3NDA5fQ.gqKmrtxKqquiPYJ3OlwGLyE9U2JnnXopV71Vi3ZAv7HO4mzmtelghZoKMPUuzPw0Ci7NSzXaWIiieOBuo3OsvyKQUbrQfajQ2OAZodsglYX9bq_eEwDeG3U3vkqMzRyWxVShNuouWmWl_oboNwSGKzTcAaDtMrGqRCWlYjTLC7s_nxyNxMjkZkrMdpZSElmCHGZFuOGmUhfMZu8rRSAXmNM-7g_dweEe6YOIr43Lm-h1aP5UIVoBwE8s63KveaVuXuIpsTixS57UFvagga0l4dkuQzBNObztfEpKDnPJt1QkUt_S2hgNhHfMUD11cQTJW0yW-ZSpl9iQg0SAsVYStg Download .mp4 (69.55 MB) Help with .mp4 files SUBMISSION 1.10.2023.mp4

Axillary artery aneurysm as rare cause of concurrent cerebellar infarct and acute limb ischemia: A case report

Axillary artery aneurysms are well documented and have potentially severe or life-threatening complications [1-3]. While pseudoaneurysms of the axillary artery can be caused by trauma or iatrogenic complications, true aneurysms are often due to repeated blunt trauma to the axillary artery over time, leading to weakening of the artery wall. The most common example is prolonged crutch use, though some athletes who have repetitive forceful movement of the upper limb, such as baseball players, may also be at risk [3]. Rarer causes include degenerative disorders such as Marfan’s syndrome or inflammatory diseases such as vasculitis [2]. Potential complications of axillary artery aneurysms include neurologic or vascular compromise of the involved upper extremity. Here we report the rare case of a patient presenting with concurrent acute cerebellar stroke and critical limb ischemia as embolic complications of axillary artery aneurysm.

Endovascular treatment of false aneurysm of axillary artery in obese women with epilepsy. 5 years follow up.

False aneurysms of arteries in upper extremities are relatively rare. They occur in 2 % of all false aneurysms, and axillary artery aneurysms are very uncommon. Open reconstructive surgery of axillary aneurysms bares the high risk of local and general complications. Progress in endovascular surgery and especially miniaturizationn of delivery systems for stents and stentgrafts enables their application in the treatment of small size, peripheral arteries. Our paper presents a case of extremely obese – 170 kg, 57 years old women, who presented with massive, false aneurysm of left axillary artery, being the consequence of epilepsy attack. She was successfully treated with endovascular deployment of covered stent Viabahn™ ø 6mm x 50mm (W.L. Gore), performed with randez-vous technique. After 5 years long follow up, the stentgraft is patent, in correct position, without radiological signs of migration or endoleaks.

Giant bilateral axillary artery aneurysms with left complete obstructive thrombus in intravenous immunoglobulin-sensitive Kawasaki disease: a case report

BackgroundKawasaki disease (KD) is a systemic vasculitis that predominantly affects medium-sized arteries. In addition to well-known coronary artery aneurysms (CAAs), peripheral systemic artery aneurysms (SAAs) have also been sporadically reported. In the literatures, SAAs occurred mainly in untreated, intravenous immunoglobin (IVIG)-resistant, or severe refractory KD, and thrombotic events in SAAs were rarely reported.Case presentationA 10-month-old boy with a history of KD was referred to our hospital for suspected pseudoaneurysm of the axillary arteries. Four months prior to presentation, he had persistent fever, conjunctival congestion, and rash. On the 10th day of fever echocardiogram showed biliteral CAAs. He was then diagnosed with KD and given IVIG 2 g/kg and aspirin at a local hospital. His fever and symptoms soon subsided and he was discharged with low dose aspirin and dipyridamole. One month prior to presentation, his parents incidentally palpated swellings in his bilateral axillae. On admission, physical examination revealed a pulsatile swelling in his right axilla and a non-pulsatile swelling in the left with impalpable left brachial and radial pulses, cooler and less active left upper limb than the right one. While the pulses of other three limbs were normal. Ultrasound examination revealed giant bilateral axillary artery aneurysms (AAAs) with massive thrombus in the left. Angiography confirmed giant bilateral AAAs with left AAAs completely occluded and fine collateral vessels connecting to the distal brachial artery, in addition to giant bilateral multiple CAAs without stenoses. The patient was given intravenous prostaglandin for 10 days to allow for formation of collateral circulation, as well as aspirin, low molecular weight heparin (which was switched to warfarin before discharge) and metoprolol. At discharge, the temperature and movement of his left upper limb improved significantly. On follow-up at 7 months, his left upper limb further improved and was similar to the right with no occurrence of cardiovascular events. The images of CAAs and AAAs on echocardiogram and computerized tomography remained the same.ConclusionsThis case highlights the importance of evaluating peripheral SAAs in KD patients with CAAs, even if their course of treatment appears smooth. For both large non-aortic SAAs and CAAs in KD patients, antithrombotic therapy is of utmost importance.

Nonoperative Management of Thrombosed Infantile Axillary Artery Aneurysm

Nonoperative Management of Thrombosed Infantile Axillary Artery Aneurysm

Neonatal Kawasaki disease with multiple arterial aneurysms: a case report

BackgroundKawasaki disease (KD) is a medium vessel vasculitis that typically occurs in children aged between 6 months and 5 years. It is extraordinarily rare in the neonatal period. KD-related systemic artery aneurysms (SAAs) have never been reported in neonates.Case presentationA male infant was transferred to our institution for persistent high-grade fever lasting 16 days. Symptoms started at day 14 of life, and he was admitted to a children’s hospital on the second day of fever. Physical examination at the time found no signs suggestive of KD. The only laboratory parameters which were of significance were values suggestive of systemic inflammation. However, his fever persisted and inflammatory markers continued to rise despite 2 weeks of antibiotic therapy. KD as a noninfectious cause of fever was considered when he came to our institution, and echocardiographic findings of left and right medium coronary artery aneurysms (CAAs) confirmed our suspicions. Full-body magnetic resonance angiography also revealed bilateral axillary artery aneurysms. Administration of intravenous gamma globulin resulted in rapid improvement. His fever resolved on the next day and CAAs and SAAs regressed to normal at 6 months and 3 months after diagnosis, respectively.ConclusionThis unique case of incomplete KD highlights the importance of considering KD in neonates with unexplained prolonged fever and reinforces the need to remain vigilant for SAAs in KD.

Substantiation of the choice of open surgical intervention in treatment of false axillary artery aneurysm

Substantiation of the choice of open surgical intervention in treatment of false axillary artery aneurysm

A rare case of a true (degenerative) axillary artery aneurysm, secondary to chronic usage of crutches and its surgical management

Aneurysms of the axillary artery are rare but potentially dangerous lesions that threaten the upper extremity with vascular and neurologic compromise. These aneurysms may arise as an aneurysm or pseudoaneurysm secondary to trauma or iatrogenic complications, or as degenerative lesions but secondary to the chronic use of crutches is rare. Inappropriate placement of the patient’s body weight on the axillary pad by the crutch causes repetitive trauma to an axillary artery leading to stenosis or aneurysm formation. The signs and symptoms vary with the cause of the aneurysm. It may include mass effects with brachial plexus compression and thromboembolic events involving the hands and fingers. Treatment should be considered in most of these lesions to prevent spontaneous rupture or microembolisation to the extremity leading to ischemia, gangrene, and amputation of the affected extremity. Surgical resection of aneurysm remains mainstay treatment followed by either interposition graft or direct end to end anastomosis.

Clinical history and management recommendations of the smooth muscle dysfunction syndrome due to ACTA2 arginine 179 alterations

PurposeSmooth muscle dysfunction syndrome (SMDS) due to heterozygous ACTA2 arginine 179 alterations is characterized by patent ductus arteriosus, vasculopathy (aneurysm and occlusive lesions), pulmonary arterial hypertension, and other complications in smooth muscle–dependent organs. We sought to define the clinical history of SMDS to develop recommendations for evaluation and management. MethodsMedical records of 33 patients with SMDS (median age 12 years) were abstracted and analyzed. ResultsAll patients had congenital mydriasis and related pupillary abnormalities at birth and presented in infancy with a patent ductus arteriosus or aortopulmonary window. Patients had cerebrovascular disease characterized by small vessel disease (hyperintense periventricular white matter lesions; 95%), intracranial artery stenosis (77%), ischemic strokes (27%), and seizures (18%). Twelve (36%) patients had thoracic aortic aneurysm repair or dissection at median age of 14 years and aortic disease was fully penetrant by the age of 25 years. Three (9%) patients had axillary artery aneurysms complicated by thromboembolic episodes. Nine patients died between the ages of 0.5 and 32 years due to aortic, pulmonary, or stroke complications, or unknown causes. ConclusionBased on these data, recommendations are provided for the surveillance and management of SMDS to help prevent early-onset life-threatening complications.

Hybrid repair of multiple subclavian and axillary artery aneurysms in a patient with Marfan syndrome

Hybrid repair of multiple subclavian and axillary artery aneurysms in a patient with Marfan syndrome

A Case of Bilateral Axillary Artery Aneurysms Caused by Fibromuscular Dysplasia

A Case of Bilateral Axillary Artery Aneurysms Caused by Fibromuscular Dysplasia

川崎病に合併した残存腋窩動脈瘤の1手術例

川崎病に合併した残存腋窩動脈瘤の1手術例