Amateur Bodybuilder Research Articles

Use of Point-of-care Ultrasound to Diagnose Rectus Abdominis Strain in the Acute Setting: A Case Report.

Rectus abdominis muscle strains are common and can be debilitating in both professional and amateur athletes who engage in strenuous activity. We report a rare case of rectus abdominis muscle tear in an amateur bodybuilder diagnosed by point-of-care ultrasound (POCUS) in the emergency department (ED). The patient had presented to the ED three separate times after strenuous exercise, received costly diagnostic workups, and ultimately was diagnosed on the third visit with grade 2 bilateral rectus abdominis tear. The patient was given appropriate education and sports medicine follow-up. He underwent rehabilitation focused on trunk and core stability. At eight-week follow-up, the patient had made a full recovery. To our knowledge, a case of bilateral rectus abdominis tear diagnosed by ultrasound in the emergency setting has not been previously published. Our case report highlights the utility of POCUS in diagnosing musculoskeletal pathology and preventing costly bounce-back visits.

Biceps brachii long head calcific tendinitis can be treated quickly and effectively with radial shockwave therapy: a case report

Calcific tendinitis, which mostly involves the supraspinatus and rotator cuff tendons, may very rarely involve the long head of the biceps brachii (LHBB). Arthroscopy is the first choice in cases of calcific tendinitis accompanied by a rotator cuff tear but in cases where the rotator cuff is intact, there are different treatment methods, such as non-steroidal anti-inflammatory drugs (NSAID), steroid injection, Extracorporeal Shock Wave Therapy (ESWT), and Radial Shock Wave Therapy (RSWT). A 28-year-old male presented at the outpatient clinic with right shoulder pain which had been ongoing for 6 months. The patient, who was a licensed table tennis player and amateur bodybuilder, had no systemic diseases. After clinical and radiological evaluation, ultrasonography showed calcification at the level of the greater tuberculum of the right humerus. The patient was diagnosed with calcific tendinitis of the bicipital tendon. Magnetic resonance imaging (MRI) was then performed to investigate any rotator cuff pathology or glenohumeral joint pathology. No additional pathology was observed. The patient was managed with the standard RSWT protocol of four sessions. 0.25 mJ/mm2 3 bar 2000 beats administered with an electromagnetic lithotriptor. Before RSWT, the VAS score was 9 over 10 and Constant score was 37, and after the first session of RSWT, the VAS score of the patient had decreased to 6. After the 4th session, treatment was terminated as the patient was completely pain-free. At the 1-year follow-up examination, the patient was still pain-free during daily activities and sports activities, and had full range of motion. In this case report, RSWT is presented as an effective treatment option for LHBB calcific tendinitis. RSWT may be a priority option over ultrasound-guided or arthroscopic barbotage in patients without tendon rupture.

Fatal anabolic androgenic steroid overdose in an amateur bodybuilder: a clinical and autopsy report.

Stanozolol is a synthetic anabolic-androgenic steroid commonly used by bodybuilders to increase muscle mass. However, its use can lead to serious adverse effects on the liver, including cholestasis, hepatic necrosis, and even death. In this case report, we describe a fatal case of stanozolol overdose in an otherwise healthy 35-year-old amateur bodybuilder. The patient presented with general malaise, jaundice, and a history of hematemesis after taking stanozolol tablets orally for 3months. Upon admission, his liver function tests were significantly abnormal, and he succumbed within 48h despite symptomatic treatment. The autopsy revealed sub-massive hepatic necrosis, focal macro-vesicular steatosis, and a cholestatic pattern of acute liver injury, with the chemical examination confirming the presence of stanozolol in the blood, liver, and kidneys. The cause of death was determined to be hepatic necrosis as a complication of stanozolol overdose. The overuse of anabolic steroids like stanozolol can cause hepatotoxicity, resulting in reversible cholestatic hepatitis or, in rare cases, fatal liver injury. The mechanism of anabolic androgenic steroid (AAS) drug-induced liver injury is obscure, but proposed mechanisms include oxidative stress and cholestasis. In this case, the recent overuse of stanozolol, a 17 alpha-alkylated (oral) AAS led to sub-massive hepatic necrosis and subsequent liver failure, proving fatal. It is imperative that healthcare providers and the public are informed about the dangers of AAS use, especially since AAS usage has increased recently due to easy online access, to prevent potentially life-threatening consequences.

Anabolic Androgenic Steroids Persistently Modify the User's Lipid Profile: a Case Report

The aim of the study was to show a lasting modification of the lipid profile and atherogenic indices by the abuse of anabolic androgenic steroids (AAS) by a amateur bodybuilder. This study reports a case of a 27-year-old non-competitive bodybuilder with severe abnormalities in lipid profile and atherogenic indices. He used anabolic androgenic steroids and had no other risk factors for heart disease or associated risk factors. After some educational lectures, he decided to continue his training without using anabolic androgenic steroids or any illegal ergogenic drugs. Lipid profile and atherogenic indices returned to normal values ​​in the third laboratory test performed five years after her last cycle of AAS. Abusing anabolic androgenic steroids can promote increased risk of cardiovascular disease, a trait that can persist for many years after even after discontinuing anabolic androgenic steroid use.

S2851 Harm by SARM: A Case of Drug-Induced Liver Injury in an Amateur Bodybuilder

S2851 Harm by SARM: A Case of Drug-Induced Liver Injury in an Amateur Bodybuilder

High Resistance Training Volume and Low Caloric and Protein Intake Are Associated with Detrimental Alterations in Body Composition of an Amateur Bodybuilder Using Anabolic Steroids: A Case Report

Many bodybuilders use anabolic androgenic steroids (AAS) to potentiate muscle mass gain as a result of specific resistance training and nutrition. The case we describe hereafter outlines alterations in body composition of an amateur bodybuilder during his pre-competitive period (11 weeks). The Participant was a 28-year old Caucasian male who was aiming to participate in his first bodybuilding competition under the “Men’s Physique” category. The Participant self-administered AAS for the whole pre-competitive period, followed a 13 set-meal plan consisting of 10 daily meals interspersed by approximately 2 h, and performed six high-volume resistance-training sessions a week. Body mass and anthropometric variables were measured at the beginning, during and at the end of his pre-competitive period. Dietetic analysis revealed a reduction in protein intake at the 10th (% decrease: −51.3%) and 11th weeks (% decrease: −64.5%) and in calorie intake at the 10th (% decrease: −57.9%) and 11th weeks (% decrease: −59.5%) in comparison to the 1st week. As a result, the Participant reduced both total body mass and fat percentage. However, there was a reduction in absolute fat free mass, despite the use of AAS. Apparently, the combination of excessive volumes of training with inadequate protein consumption was responsible for this outcome.

Bodybuilders pneumonia.

Bodybuilders pneumonia.

Caution for Anabolic Androgenic Steroid Use: A Case Report of Multiple Organ Dysfunction Syndrome

We report a 42-year-old male amateur body builder and user of anabolic androgenic steroids, who developed ARDS, acute kidney injury, and refractory supraventricular tachycardia. He required extracorporeal membrane oxygenation, continuous veno-venous hemodialysis, and catheter ablation. We believe that long-term anabolic androgenic steroid abuse predisposed the patient to multiple organ dysfunction syndrome, from its immunomodulatory effects in an otherwise healthy patient. Anabolic androgenic steroid use should be part of the history taking process, since it may complicate diagnosis, disease progression, and prognosis.

A case of necrotizing myopathy with proximal weakness and cardiomyopathy

A male amateur body builder presented at age 23 years with a 2-year history indicative of progressive proximal limb weakness. His initial symptom was a rash which was nonpruritic and he described as “ring” lesions on his trunk which spontaneously resolved after 6 months. Following resolution of

Trenbolone Acetate Use Causing Acute Hepatitis

Purpose: Athletes continuously seek ways to improve performance in their sport and can be drawn to the use of anabolic steroids in order to obtain this goal. They tend to ignore the potentially life-threatening side effects of these drugs and consequently put their overall physical well-being at risk. We describe a case of an amateur body builder who took trenbolone acetate and subsequently developed acute hepatitis. Case: A 40-year-old male amateur bodybuilder took trenbolone acetate by injection for one year and developed jaundice. He had a seven-week history of generalized fatigue, diarrhea with light colored stools, dark urine, and right upper quadrant pain. He denied alcohol use or family history of liver disease. Physical examination showed scleral icterus with a palpable liver 3 centimeters below the costal margin. Laboratory results were significant for Aspartate aminotransferase of 121 IU/L, alanine aminotransferase of 199 IU/L, total bilirubin of 17.4 mg/dL, direct bilirubin of 9.9 mg/dL, albumin of 2.8 mg/dL, total cholesterol of 508 mg/dL, LDL of 389 mg/dL, and HDL of 10 mg/dL. Coagulation profile was normal. Ultrasonography showed a contracted gallbladder with thickened walls and no evidence of intra- or extra-hepatic ductal dilatation. Liver biopsy showed moderately severe parenchymal and canalicular cholestasis with minimal portal inflammation consistent with drug-induced cholestatic hepatitis. Discussion: Hepatic complications associated with anabolic steroid use include hepatocellular and intrahepatic cholestasis. Aminotransferases are usually less than 100 IU\L and serum alkaline phosphatase levels may be mildly elevated. HDL can be decreased and LDL can be increased, which can put these patients at risk for cardiovascular disease. Hepatocellular adenomas and peliosis hepatis have been described. Most of these effects can be reversed upon cessation of the steroids; however there have been fatalities. By methylating testosterone, it undergoes less first pass hepatic metabolism and it's oral bioavailability improves. Trenbolone acetate is a steroid approved for use in veterinary practice as a means of maintaining cattle weight in the days prior to slaughter. It is not approved, nor studied in humans, and its hepatic side effect profile is unknown. Our case reminds health care professionals about the abuse potential of anabolic steroids and the systemic side effects they can cause.

Rhabdomyolysis of the Deltoid Muscle in a Bodybuilder Using Anabolic-Androgenic Steroids: A Case Report

Rhabdomyolysis (breakdown of skeletal muscle tissue) may be caused by mechanical, physical, chemical, or biological factors. We present the unique case of a bodybuilder who developed localized rhabdomyolysis of the deltoid muscle after injection of steroids into the shoulder region. A 39-year-old amateur bodybuilder presented to the emergency department with excruciating pain and inability to move his right shoulder after injecting stanozolol, an anabolic-androgenic steroid (AAS), into his right deltoid muscle on the same day. On physical examination, the right deltoid muscle was swollen and tense and the surrounding skin red, tender, and warm. He had no fluctuation or systemic fever and no sensory or motor deficit. His distal pulsations were distinct. Laboratory test results suggested massive rhabdomyolysis. The major magnetic resonance imaging finding was diffuse hyperintensity signals on T2-weighted images of the deltoid muscle, which was consistent with edema. Polymyositis and dermatomyositis, mild injury, infectious myositis without phlegmon or abscess formation, radiation therapy, subacute denervation, compartment syndrome, early myositis ossificans, rhabdomyolysis, and sickle cell crisis. The patient was treated with intravenous fluid replacement and sodium bicarbonate to alkalinize the urine. Four days after admission, his pain had decreased, he had regained range of motion, and his renal function remained unaffected. Anabolic-androgenic steroid use is associated with various side effects that are generally systemic and dose related. We could not find reports of localized side effects of AAS use, as this case presented, elsewhere in the English-language literature. "Doping" among amateur athletes occurs frequently. It can cause acute and chronic health problems, most of which are systemic. This is the first description of localized rhabdomyolysis in the area of an AAS injection.

The dire consequences of doping

Various cases in professional sports have brought the matter of doping—the use of performance-enhancing drugs—to the attention of the broad public. Here, we present a case of doping in a 21-year-old amateur bodybuilder. The man presented to our clinic feeling generally unwell. He was subfebrile and had massive, deep ulcerations, abscesses, and pustules located on his chest and upper back (figure). Persistent questioning revealed a history of continuous abuse of anabolic-androgenic steroids (testosterone enantate 250 mg plus metandienona 30 mg twice weekly), which was consistent with the diagnosis of a severe acne conglobata that was anabolic-androgenic steroid induced.

Caffeine toxicity in a bodybuilder.

Substance abuse is well documented among bodybuilders and weight lifters keen to enhance their performance. A case is described of abuse of caffeine to toxic levels by an amateur bodybuilder...