patient haddefinite fatty infiltration of theRV. Incidentally CMR detected important previously undiagnosed pathology including anomalous pulmonary venous drainage (2 patients) andnon-ischaemic cardiomyopathy (6.5%).CMR was normal in 63%, with minor abnormalities in 28.5%. Conclusions: CMR is an important diagnostic tool in the evaluation of possible ARVC. Patients who do not meet TFC for diagnosis may have CMR features typical of ARVC. Additionally CMRmay detect other hitherto undiagnosed structural or functional abnormalities that may alter patient management. doi:10.1016/j.hlc.2008.05.073 73 Measurement of Temporal Heterogeneity as a Marker of Ischemia at Exercise Stress Echocardiography:Use ofContraction Front Mapping of Real-time 3D Echo Carly Jenkins ∗, Brian Haluska, Thomas H. Marwick University of QLD, Brisbane, Australia 2DESE (2DE) is limited by standard imaging planes because of poor matching stress images. Real-time 3D ESE (RT3D) may avoid these orientation problems and contraction front mapping (CFM) provides a method for defining temporal homogeneity of contraction.We sought the incremental benefit of RT3D CFM to 2DE and RT3D, 74 Hand-HeldUltrasound isComparable toLab-BasedUltrasound for the Assessment of Carotid Intima–media Thickness: A New Tool to Increase Cardiovascular Risk Assessment Brian A. Haluska ∗, Joseph Brown, Thomas H. Marwick University of Queensland, Brisbane, Qld, Australia Background: Hand-held ultrasound (HH) has been increasingly used and shown to be comparable to larger, lab-based machines (LB) in assessing LV and valvular function. However, use of HH for assessment of vascular function has not been widely explored. We sought to compare HH with LB for the measurement of carotid intima–media thickness (IMT), an important marker of preclinical atherosclerosis. Methods: We studied 33 pts (21 men; age 42± 14) with and without CVD. The common carotid arteries (CCA) were scanned by 2 experienced sonographers using HH (Vivid I; GEUltrasound) and LB (IE33; PhilipsUltrasound) machines, both with 11MHZ linear probes and similar frame rates (40–45 FPS). IMT was measured off-line in the far wall for both HH and LB by one observer using a fixed-size ROI. Pearson’s correlation, paired t-tests and Bland–Altman analysis was then performed. Results: Correlation was high between both machines for IMT: LCCA (r= 0.96), RCCA (r= 0.96) and mean IMT using coronary angio as the ref. std. Methods: 70 pts (47men, 61± 10 years) underwent ESE and angio. All had 2DE (5 views) and RT3D (2 full volumes) at rest and peak stress. CFM of RT3D images was performed offline. Qualitative CFM was performed by identifyingabnormality on thedelayed regional relaxation corresponding to a coronary territory. Quantitative CFM wasbasedon thedifference in regional andglobal contraction times (CT). Accuracy of CFM defined by comparison with angio. Results: Pts were excluded for non-diagnostic stress (i.e. <80% predicted HR n= 9) or poor quality RT3D (n= 9). ROC analysis was used to define normal cutoff ranges of CT for coronary territories LAD (CT 12%), LCx (CT 10%), RCA (CT 11%) and overall (CT 12%). Table shows comparison between wall motion scoring of 2DE, RT3D and CFM to angio. Visual assessment of RT3D has a lower sensitivity than 2DE and CFM, which may reflect its lower image resolution. While the low specificity of 2DE may reflect referral bias, the higher specificity of RT3Dmay represent avoidance of false positives from off-axis imaging. Conclusions:Analysis of the temporal distribution of contraction may be a better means than 3D wall motion assessment for identification of ischemia at ESE.