Abstract
Abstract Sensitivity of burnet mitochondria to cyanide was studied and some detoxifying mechanisms were investigated. Results showed that burnet mitochondria are relatively sensitive to KCN and that the inhibitory effects of cyanide can be completely reversed by pyruvate. Additionally, some glycolytic intermediates were determined in vivo at differential times from the beginning of the crushing‐induced thanatosis and at subsequent arousal. On the basis of our results, a hypothesis is suggested to explain both the well‐known resistance shown in vivo by burnets to hydrocyanic acid, and the biochemical mechanism at the root of thanatosis.
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