Abstract
For most of its centuries-old history, acute pneumonia (AP) has been considered by medicine as a single nosological form based on inflammation of the lung tissue and which had individual differences in severity and aggressiveness of its development. The lack of any information about the causative agents of the disease did not pose the task of searching for etiotropic agents, therefore, medical care for such patients was carried out for a long time mainly using pathogenetic methods found empirically.
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