Abstract
Myasthenia gravis (MG) is a disease with autoimmune etiology. The main pathomechanism is related to the production of antibodies against nicotinic acetylcholine receptor (AChR) located in the postsynaptic membrane. The present study is set to compare the serum level of interleukin-33 (IL-33)/receptor ST2 in patients with myasthenia gravis and controls; and to study the relation with the severity of disease. Our observations confirm that a significant increase in serum concentrations of IL-33 and ST2 was observed in MG patients compared to healthy individuals. This relation is not precisely known yet, which requires more studies.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
