Abstract
Aim: Acute kidney injury (AKI) occurs in up to 20% of adult patients after cardiac surgery. We have shown in previous human studies that persistently low intraoperative urinary oxygen tension (pO2) is correlated with a high risk of AKI. However, before attempting to intervene in humans, by altering the cardiopulmonary bypass (CPB) variables (flow, pressure, haematocrit, and temperature), it is necessary to determine the most effective methods to improve oxygenation in an animal model.
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