Abstract
Posterior cortical atrophy (PCA) is a dementing syndrome that presents with signs and symptoms of cortical visual dysfunction.1 The clinical features of PCA reflect dysfunction of both the ventral/occipito-temporal visual processing pathway causing apperceptive visual agnosia, alexia, and prosopagnosia and dorsal/occipito-parietal pathway causing Balint syndrome (simultanagnosia, optic ataxia and ocular apraxia), transcortical sensory aphasia, apraxia, and some or all of the elements of Gerstmann syndrome (agraphia, acalculia, finger agnosia, right-left disorientation).2,3 Frontal lobe functions and memory are relatively preserved until later in the course of the disease. In this issue of Neurology , McMonagle et al.4 describe the cognitive profile of 19 patients with PCA and compare this to the profile of 11 patients with typical amnestic Alzheimer disease …
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