Abstract
Patients infected with the human immunodeficiency virus type I (HIV-I) suffer from numerous neurological complications related to immunodeficiency. Neurological symptoms are seen in 10 to 25% of all patients with clinically manifest acquired immunodeficiency syndrome (AIDS) or AIDS-related complex (ARC). Over 50% of all patients develop encephalopathy disorders in the terminal stage of the disease [1]. Electrophysiologically recorded changes of the central nervous system (CNS) have also been described in healthy HIV-I-seropositive patients without clinically apparent symptoms [2]. HIV-I was demonstrated in grey and white matter, in neurons and glia cells and was isolated from cerebrospinal fluid (CSF) [1,3].
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