Abstract
Prolongation of QT associated with electrolyte changes can lead to ventricular arrhythmias. Correction and supply of calcium, magnesium, and potassium are essential to managing this condition. In this report, we present a case of QT prolongation due to magnesium and vitamin D deficiency in a patient with rheumatoid arthritis.
Highlights
Glucocorticoids are routinely used to treat various diseases
Prolonged glucocorticoid use is associated with hypocalcemia as well as magnesium and vitamin D deficiencies [1,2], and vitamin D and magnesium deficiencies cause hypocalcemia
Hypocalcemia and magnesium deficiency can result in QT interval prolongation, predisposing the patient to life-threatening ventricular arrhythmias [3,4]
Summary
Glucocorticoids are routinely used to treat various diseases. One of the main indications for glucocorticoid therapy is inflammatory rheumatic disorders. A 60-year-old man presented to the emergency department of our institution with palpitations and an altered mental state due to ventricular tachycardia His medical history included pneumoconiosis, nontuberculous mycobacteria lung disease, chronic obstructive lung disease, heart failure, and rheumatoid arthritis. Laboratory test results showed a pro B-type natriuretic peptide level of 3,315 pg/mL (reference range:
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