Abstract
Venous CO2 loading was achieved in unanesthetized, spontaneously breathing white Pekin ducks by gut-ventilation to load CO2 across the intestinal circulation. Gut ventilation with air had no effect on breathing, while venous CO2 loading resulted in an increase in minute ventilation [Formula: see text]. Ventilation sensitivity [Formula: see text] when expressed as a percentage of [Formula: see text] obtained either without or during gut ventilation with air (15%∙mmHg−1; 1 mmHg = 133.322 Pa) was within the range of values previously recorded in birds by investigators using other techniques. An isocapnic hyperpnea, as observed in some other studies, was never seen using gut loading of CO2. These experiments have shown the efficacy of loading CO2 across the gut wall in ducks and this technique should prove useful in resolving the roles of the various CO2-sensitive receptor groups in increasing [Formula: see text] in response to CO2 loads.
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