Abstract
Signaling through the serum and glucocorticoid inducible kinase-1 (SGK-1) can be mechanically activated. The objective of this investigation was to demonstrate that in vitro cyclic stretch of aortic vascular smooth muscle cells (VSMCs) and in vivo induction of hypertension (HTN) can cause SGK-1-dependent production of cytokines to promote macrophage accumulation in the murine abdominal aorta.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
