Abstract
The high degree of hepatitis B virus (HBV) quasispecies diversity in chronically infected individuals may suggest that viral variants favouring resistance to specific nucleot(s)ide analogues (NA) may pre-exist to antiviral treatments. Aim of this study was to investigate the genetic variability of the entire HBV reverse transcriptase (RT) domain and of the overlapping S gene in a large series of both untreated and lamivudine-resistant patients.
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