Abstract
Beta-adrenergic receptor stimulation increases calcium influx through the pore forming alpha subunit of the cardiac L-type Ca2+ channel (CaV1.2). Chronic activation of the CaV1.2 can lead to development of cardiac hypertrophy and arrhythmia formation; yet the molecular mechanism remains unresolved.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
