Abstract
THROMBOCYTOPENIA may result from impaired production of platelets, increased destruction of platelets or a combination of these factors. At the present time this etiologic distinction is generally made on the basis of the number and appearance of the megakaryocytes in the bone marrow. Rarely, a more exact classification can be made only after performance of platelet-survival studies.1 Platelets are rich in the enzyme acid phosphatase,2 3 4 5 and therefore their excessive destruction in vivo might lead to elevations in the plasma levels of this enzyme. In a group of patients with thrombocytopenia from a variety of causes the assay of acid phosphatase . . .
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