Abstract
There has been considerable debate regarding the effect of vitamin C on the prevention of complex regional pain syndrome (CRPS) in the setting of distal radius fractures.1–3 Vitamin C, also known as ascorbic acid, is a water-soluble organic compound first identified in 1932. Although humans are unable to synthesize vitamin C, it is an essential micronutrient in many enzymatic and chemical pathways. Vitamin C acts as a cofactor for 8 different enzymes that are involved in collagen hydroxylation, carnitine and norepinephrine biosynthesis, amidation of peptide hormones, and tyrosine metabolism.
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