Abstract
Background: Mandibles that present 5 mm or less at their most atrophic point are at high risk for spontaneous fractures. This change in bone volume after tooth loss results in decreased vascular supply from the endosteal and periosteum, as well as decreased potential for osteogenesis, which may compromise bone healing in this population group. The current treatment for atrophic mandible is commonly separated into closed reduction, external fixation and rigid internal fixation. Many surgeons have proposed the use of bone graft at the time of fracture treatment as a way to increase the probability of bone fusion.
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