Abstract
The urinary albumin excretion of a group of 96 subjects with strong hereditary predisposition to diabetes was compared with that of 41 non-diabetics and 59 established diabetics by a sensitive radioimmunoassay method. Only in the established diabetics was urinary albumin loss significantly raised. This supports the view that diabetic glomerulosclerosis is a consequence rather than a forerunner of the clinical disease.
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