Abstract

Cutaneous leishmaniasis (CL) is one of the most endemic and neglected diseases worldwide, still representing an important public health problem. Its clinical presentations are very polymorphic and sometimes very difficult to diagnose. Among the variants described as “atypical” or “unusual”, eyelids CL is exceptional with a prevalence estimated at only 0.27-0.72% in large series. In most cases, eyelids leishmaniasis is skin-limited disease, but can exceptionally cause serious ocular complications and may lead to blindness. We present an original case of isolated upper left eyelid CL in a 28-years-old Tunisian man. As rare as it is, this atypical clinical presentation of CL deserves to be well known by clinicians, and discussed in front of any palpebral lesion which does not prove itself in patient living or returning from a country endemic for this infection.

Highlights

  • Cutaneous leishmaniasis (CL) is a cosmopolitan parasitic infection [1,2,3,4]

  • The clinical presentation of this parasitosis is characterized by a large polymorphism [1,2,6] with so-called “atypical” and “unusual” forms [2,6,7]

  • We present an original case of isolated upper left eyelid CL in a 28-years-old Tunisian man

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Summary

Introduction

Cutaneous leishmaniasis (CL) is a cosmopolitan parasitic infection [1,2,3,4]. it remains among the most neglected diseases and represents a real public health problem in several countries of the world [2,4], its incidence is estimated at two million cases annually [2]. The following forms of unusual/atypical CL have been described: lupoid, sporotricoid, eczematiform, verrucous, dry, zosteriform, erysipeloid, psoriasiform, pseudotumoral, discoid lupus erythematosus-like, eyelids, squamous cell carcinoma-like, erythematous volcanic ulcer, paronychial, chancriform, and annular forms [2,6,7]. These forms are very challenging for clinicians, those in the first line and in nonendemic areas [2]. Nodular, crusted, 2 cm × 1 cm in diameter, and painless skin lesion located in upper left eyelid (Figure 1&2) without involvement of anterior eye structures (Figure 3).

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