Abstract
Hypertrophic obstructive cardiomyopathy (HOCM) in association with aortic disease is uncommon. However, left ventricular outflow tract obstruction (LVOTO) generates turbulent flow patterns that could augment the aneurysmal change in the aortic root in cases of connective tissue disorder.1 Phenotypic heterogeneity in HOCM cases requires a tailored approach to correct all factors contributing to the dynamic LVOTO including the mitral valve and its subvalvular apparatus.2
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