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Abstract
The expression of type III hyperlipidemia (HLP) is the topic of the paper by Peter Henneman and his colleagues.1 Only very few lipid disorders seem to be more complex and poorly understood than the type III HLP by Fredrickson (or so-called dysbetalipoproteinemia). This is true despite the fact that type III HLP had been described in great detail more than 33 years ago2 and since then had been studied intensively by the most dedicated, leading lipid researchers worldwide. Type III HLP is a genetic disorder characterized by the accumulation of remnant lipoproteins in the plasma and development of premature atherosclerosis.
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