Abstract
1. Direct application of serotonin (5-HT) or weak concentrations (10 −7 M) of acetylcholine (ACh) to the cerebral ganglion of the mussel, Mytilus edulis produced a marked increase in electrical activity recorded from cells of the visceral ganglion. 2. Exogenous application of dopamine (DA) had the opposite effect. 3. Application of phenoxybenzamine (PBZ) to the cerebral ganglion prior to ACh prevented activation of visceral ganglion cells and activation of ciliary beating. 4. A model is presented to account for the coordinated control of ciliary beating by the cerebral and visceral ganglia.
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