Abstract
Aims: Heart failure may be due to a lack of ATP production. Coordinated downregulation of the key regulator of mitochondrial ATP production, PGC-1α has been implicated in the development of heart failure (Garnier et al. J Physiol. 2003). We aimed to assess the expression of PGC-1α and respiratory chain genes as well as contractile activity during the development of pressure-overload hypertrophy and heart failure.
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