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Abstract
It is shown that regenerative plastic insufficiency develops in cardiomyocytes of young and old rats after a one-time general overheating. Atrophic and necrobiotic alterations of the cardiomyocytes together with a decrease of the heart weight are noted as well. Intracellular alterations of cardiomyocytes of young and old rats are accompanied by a marked decrease of succinate dehydrogenase activity under these conditions. There-after its activity rises appreciably in young animals during the course of postheating restitution, while in old rats the activity of this enzyme remains low.
Published Version
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