Abstract
A 47-year-old woman with a prior medical history of SLE and chronic arterial hypertension developed a post-anoxic coma after a prolonged cardiac arrest. She also presented a convulsive status epilepticus.
Highlights
A 47-year-old woman with a prior medical history of SLE and chronic arterial hypertension developed a post-anoxic coma after a prolonged cardiac arrest
Cortical laminar necrosis is a specific type of cortical infarction, which usually develops as a result of generalized hypoxia rather than a local vascular abnormality
Depletion of oxygen or glucose as in anoxia, hypoglycemia, status epilepticus, ischemic stroke and less common in immunosuppressive therapy and polychemotherapy has been attributed as an underlying cause of cortical laminar necrosis
Summary
A 47-year-old woman with a prior medical history of SLE and chronic arterial hypertension developed a post-anoxic coma after a prolonged cardiac arrest. Typical CT and MRI features of cortical laminar necrosis At admission contrast enhanced CT brain (not illustrated) showed minimal loss of gray-white differentiation in the left sylvian region. This examination showed hyperdensity in a laminar way, following the cortical gyri, mostly in the temporoparieto-occipital region (Fig. A and B).
Sign-in/Register to view highlights & summary Sign-in/Register to access full text options 
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
