Abstract
Rats with lesions of the ventromedial hypothalamus performed a two-way active avoidance task significantly better than control animals, or animals with lesions extending outside the VMH. This facilitation was not a function of shock intensity; animals with VMH lesions performed identically regardless of shock level. Control rats performed better at low intensities than at high intensities. The results are consistent with the hypothesis that VMH lesions impair the normal tendency to inhibit the return to the previously shocked compartment.
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