Abstract
Background and Aims: Our previous studies have demonstrated altered pro-inflammatory activity of circulating monocytes in patients with atherosclerosis. Moreover, atherosclerosis progression and monocyte pro-inflammatory activity were associated with mitochondrial DNA (mtDNA) mutations in circulating monocytes. It's well recognized that mitochondria can act as immunomodulators through releasing molecules associated with bacterial infection. We hypothesized that atherosclerosis can be associated with changes in the mitochondrial function in circulating monocytes.
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