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Abstract
After radiofrequency ablation (RFA) of Barrett's esophagus (BE), the epithelium reverts to neosquamous epithelium (NSE). However, NSE shows characteristics of defective barrier function (increased paracellular permeability and dilated intercellular spaces) similar to those seen in gastroesophageal reflux disease (GERD). These defects which may reflect exposure of NSE to refluxed acid, and may be a risk factor for recurrence of BE. Since defective barrier function in GERD involves cleavage of the junctional protein e-cadherin, we considered the possibility that a similar phenomenon also occurs in NSE.
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