Abstract
There is much circumstantial evidence that excessive vagal activity is an etiologic factor in the majority of chronic duodenal ulcers — hence the popularity of vagotomy as a rational operation for this disorder. Although adequate vagotomy allows most ulcers to heal, and reduces gastric secretion, it does not necessarily follow that excessive vagal stimulation has been directly responsible for the ulcer in the first place. It is well known that vagal denervation not only abolishes the interdigestive secretion of the stomach but also reduces the responsiveness of the gastric secretory cells to various stimuli, and it therefore is also theoretically . . .
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