Abstract
When griseofulvin (Fulvicin) was made available for the treatment of fungus diseases, there was some question whether it was fungistatic or fungicidal. This became evident when fungus infections of the finger or toe nails were treated.1The growth was apparently inhibited by continuous treatment for a number of months, while an old nail was replaced by a new one. If treatment was discontinued before the new nail had completely appeared, the fungus would start growing again and the prior condition would return. Since inflammation accompanying some fungus diseases of the skin1subsides rather abruptly, it was thought possible by us that griseofulvin might be another anti-inflammatory agent. Proceeding on this assumption, we decided to administer the drug to some of our patients with rheumatoid arthritis. Among the group of patients were a few with cases of shoulder-hand syndrome. The response in patients with rheumatoid arthritis was negative, while
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