Abstract
Background and Aims : Pulmonary arterial hypertension (PAH), especially associated with congenital heart disease (CHD) of left-to-right shunt, is a big problem in medicine in terms of diagnostics and treatment. It evolves through increase of vasculare resistance and/or increase of blood flow through lungs, where the mean pulmonary artery pressure is above 25 mmHg and during catheterization above 30 mmHg. Due to volume and pressure load of blood from the left heart cavities in right, hypertrophy and insufficiency with increased production of PAH mediators, necrotizing arteritis of pulmonary capillaries occurs.
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